Endogenous CCK and the peripheral neural substrates of intestinal satiety. - Wikidata - awgldk - TriplyDB

Endogenous CCK and the peripheral neural substrates of intestinal satiety.

Endogenous CCK and the peripheral neural substrates of intestinal satiety.

http://www.wikidata.org/entity/Q40753298

about

Upper gastrointestinal dysmotility after spinal cord injury: is diminished vagal sensory processing one culprit?c-Fos generation in the dorsal vagal complex after systemic endotoxin is not dependent on the vagus nerve.Experimental spinal cord injury in rats diminishes vagally-mediated gastric responses to cholecystokinin-8s.Orexins control intestinal glucose transport by distinct neuronal, endocrine, and direct epithelial pathways.Neuroendocrine and physiological regulation of intake with particular reference to domesticated ruminant animals.Duodenal loading with glucose induces fos expression in rat brain: selective blockade by devazepide.Enterostatin inhibition of dietary fat intake is dependent on CCK-A receptors.Increased food intake and CCK receptor antagonists: beyond abdominal vagal afferents.Endogenous cholecystokinin reduces food intake and increases Fos-like immunoreactivity in the dorsal vagal complex but not in the myenteric plexus by CCK1 receptor in the adult rat.Development of an ex Vivo Method for Multi-unit Recording of Microbiota-Colonic-Neural Signaling in Real Time.

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P2860

Endogenous CCK and the peripheral neural substrates of intestinal satiety.

http://www.wikidata.org/entity/Q40753298

description

1994 nî lūn-bûn

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1994年の論文

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1994年論文

@yue

1994年論文

@zh-hant

1994年論文

@zh-hk

1994年論文

@zh-mo

1994年論文

@zh-tw

1994年论文

@wuu

1994年论文

@zh

1994年论文

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name

Endogenous CCK and the peripheral neural substrates of intestinal satiety.

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type

label

Endogenous CCK and the peripheral neural substrates of intestinal satiety.

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prefLabel

Endogenous CCK and the peripheral neural substrates of intestinal satiety.

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J.1749-6632.1994.TB44073.X

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Annals of the New York Academy of Sciences

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Endogenous CCK and the peripheral neural substrates of intestinal satiety.

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P2093

Brenner LA

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Ritter RC

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Tamura CS

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P2860

Brain and gastrointestinal cholecystokinin receptor family: structure and functional expression

Differentiation of central cholecystokinin receptor binding sites using the non-peptide antagonists MK-329 and L-365,260.

Distribution patterns of CCK and CCK mRNA in some neuronal and non-neuronal tissues.

The site of action of capsaicin on the guinea-pig isolated ileum.

Local effector functions of capsaicin-sensitive sensory nerve endings: involvement of tachykinins, calcitonin gene-related peptide and other neuropeptides.

Localization and molecular heterogeneity of cholecystokinin in the central and peripheral nervous system.

Capsaicin-induced neuronal degeneration: silver impregnation of cell bodies, axons, and terminals in the central nervous system of the adult rat.

The cholecystokinin antagonist, proglumide, increases food intake in the rat.

Capsaicin-sensitive nerves modulate resting blood flow and vascular tone in rat gut.

Intestinal peristalsis associated with release of immunoreactive substance P.

P304

255-267

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scholarly article

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10.1111/J.1749-6632.1994.TB44073.X

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713

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8185167

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