Severe NAFLD with hepatic necroinflammatory changes in mice fed trans fats and a high-fructose corn syrup equivalent. - Wikidata - awgldk - TriplyDB

Severe NAFLD with hepatic necroinflammatory changes in mice fed trans fats and a high-fructose corn syrup equivalent.

Severe NAFLD with hepatic necroinflammatory changes in mice fed trans fats and a high-fructose corn syrup equivalent.

http://www.wikidata.org/entity/Q40762653

about

Utilization of animal models to investigate non-alcoholic steatohepatitis-associated hepatocellular carcinoma Animal Models of Nonalcoholic Steatohepatitis: Eat, Delete, and Inflame Animal Models for Fibrotic Liver Diseases: What We Have, What We Need, and What Is under Development Omega-3 fatty acids for the treatment of non-alcoholic fatty liver disease Carbohydrate intake and nonalcoholic fatty liver disease: fructose as a weapon of mass destruction Low-carbohydrate ketogenic diets, glucose homeostasis, and nonalcoholic fatty liver disease Metabolic factors and chronic hepatitis C: a complex interplay Fructose as a key player in the development of fatty liver disease Role of mitochondria in nonalcoholic fatty liver disease Long term metabolic syndrome induced by a high fat high fructose diet leads to minimal renal injury in C57BL/6 mice Combination of alcohol and fructose exacerbates metabolic imbalance in terms of hepatic damage, dyslipidemia, and insulin resistance in rats The Selective SGLT2 Inhibitor Ipragliflozin Has a Therapeutic Effect on Nonalcoholic Steatohepatitis in Mice Lipotoxicity in the liver.Progression of non-alcoholic steatosis to steatohepatitis and fibrosis parallels cumulative accumulation of danger signals that promote inflammation and liver tumors in a high fat-cholesterol-sugar diet model in mice.4Ps medicine of the fatty liver: the research model of predictive, preventive, personalized and participatory medicine-recommendations for facing obesity, fatty liver and fibrosis epidemics.Fructose-Rich Diet Affects Mitochondrial DNA Damage and Repair in Rats.Fast food diet with CCl4 micro-dose induced hepatic-fibrosis--a novel animal model.Evolving concepts in the pathogenesis of NASH: beyond steatosis and inflammation.Specific Macronutrients Exert Unique Influences on the Adipose-Liver Axis to Promote Hepatic Steatosis in Mice.Translational approaches: from fatty liver to non-alcoholic steatohepatitis.High-fructose, medium chain trans fat diet induces liver fibrosis and elevates plasma coenzyme Q9 in a novel murine model of obesity and nonalcoholic steatohepatitis.GLP-1 analogs reduce hepatocyte steatosis and improve survival by enhancing the unfolded protein response and promoting macroautophagy.Modeling progressive non-alcoholic fatty liver disease in the laboratory mouse.Emodin prevents intrahepatic fat accumulation, inflammation and redox status imbalance during diet-induced hepatosteatosis in rats.Toward a unifying hypothesis of metabolic syndrome.Loss of 5α-reductase type 1 accelerates the development of hepatic steatosis but protects against hepatocellular carcinoma in male mice.Age-dependent effect of high-fructose and high-fat diets on lipid metabolism and lipid accumulation in liver and kidney of rats.Time-restricted feeding is a preventative and therapeutic intervention against diverse nutritional challenges.Fructose impairs glucose-induced hepatic triglyceride synthesis.Inflammatory Signals shift from adipose to liver during high fat feeding and influence the development of steatohepatitis in mice From whole body to cellular models of hepatic triglyceride metabolism: man has got to know his limitations Metabonomic investigations of age- and batch-related variations in female NMRI mice using proton nuclear magnetic resonance spectroscopy.Non-alcoholic fatty liver disease and fructose: bad for us, better for mice.Vascular adhesion protein-1 promotes liver inflammation and drives hepatic fibrosis A mechanism by which dietary trans fats cause atherosclerosis.Nutrition and physical activity in NAFLD: an overview of the epidemiological evidence.RETRACTED: Hepatic glucagon action is essential for exercise-induced reversal of mouse fatty liver.Inhibition of aldose reductase activates hepatic peroxisome proliferator-activated receptor-α and ameliorates hepatosteatosis in diabetic db/db mice.Fast food diet mouse: novel small animal model of NASH with ballooning, progressive fibrosis, and high physiological fidelity to the human condition.Chronic inflammation aggravates metabolic disorders of hepatic fatty acids in high-fat diet-induced obese mice.

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P2860

Severe NAFLD with hepatic necroinflammatory changes in mice fed trans fats and a high-fructose corn syrup equivalent.

http://www.wikidata.org/entity/Q40762653

description

2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年學術文章

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2008年學術文章

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2008年學術文章

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American Journal of Physiology - Gastrointestinal and Liver Physiology

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Brent A Neuschwander-Tetri

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Elizabeth M Brunt

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Laura H Tetri

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Lisa M Yerian

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Metin Basaranoglu

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P2860

Mechanism by which hyperglycemia inhibits hepatic glucose production in conscious rats. Implications for the pathophysiology of fasting hyperglycemia in diabetes.

Quantitative insulin sensitivity check index: a simple, accurate method for assessing insulin sensitivity in humans.

Fructose, weight gain, and the insulin resistance syndrome.

Preventing childhood obesity by reducing consumption of carbonated drinks: cluster randomised controlled trial.

Animal models of steatosis.

Effect of dietary trans fatty acids on the delta 5, delta 6 and delta 9 desaturases of rat liver microsomes in vivo.

Protection against fatty liver but normal adipogenesis in mice lacking adipose differentiation-related protein.

Dietary fructose reduces circulating insulin and leptin, attenuates postprandial suppression of ghrelin, and increases triglycerides in women.

Trans fatty acids and cardiovascular disease.

A trans world journey.

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