Salmonella pathogenicity island 2-dependent macrophage death is mediated in part by the host cysteine protease caspase-1.
about
Apoptosis, pyroptosis, and necrosis: mechanistic description of dead and dying eukaryotic cells.The Salmonella SPI2 effector SseI mediates long-term systemic infection by modulating host cell migrationPyroptosis: host cell death and inflammationActivation of focal adhesion kinase by Salmonella suppresses autophagy via an Akt/mTOR signaling pathway and promotes bacterial survival in macrophages.Molecular insights into farm animal and zoonotic Salmonella infections.Induction and relaxation dynamics of the regulatory network controlling the type III secretion system encoded within Salmonella pathogenicity island 1.Abundant secretion of bioactive interleukin-1beta by human macrophages induced by Actinobacillus actinomycetemcomitans leukotoxinRedundant roles for inflammasome receptors NLRP3 and NLRC4 in host defense against SalmonellaIdentification and characterization of a novel bacterial virulence factor that shares homology with mammalian Toll/interleukin-1 receptor family proteinsPolynucleotide phosphorylase negatively controls spv virulence gene expression in Salmonella enterica.Oxidized low density lipoprotein induced caspase-1 mediated pyroptotic cell death in macrophages: implication in lesion instability?Autophagy and inflammatory cell death, partners of innate immunity.Oxidative metabolism enables Salmonella evasion of the NLRP3 inflammasome.Functions and effectors of the Salmonella pathogenicity island 2 type III secretion system.The flagellar regulator TviA reduces pyroptosis by Salmonella enterica serovar TyphiRedox regulation of NLRP3 inflammasomes: ROS as trigger or effector?Salmonella enterica serovar Typhimurium requires nonsterol precursors of the cholesterol biosynthetic pathway for intracellular proliferationSseL, a Salmonella deubiquitinase required for macrophage killing and virulence.Salmonella-induced macrophage death: multiple mechanisms, different outcomes.Carbon metabolism of intracellular bacteria.Capsule-mediated immune evasion: a new hypothesis explaining aspects of typhoid fever pathogenesisMacrophage cell death upon intracellular bacterial infection.Salmonella typhimurium persists within macrophages in the mesenteric lymph nodes of chronically infected Nramp1+/+ mice and can be reactivated by IFNgamma neutralizationHow to outwit the enemy: dendritic cells face Salmonella.Roles of heat-shock protein 70 in protecting against intestinal mucosal damageInduction of guanylate binding protein 5 by gamma interferon increases susceptibility to Salmonella enterica serovar Typhimurium-induced pyroptosis in RAW 264.7 cells.Anthrax lethal toxin-induced inflammasome formation and caspase-1 activation are late events dependent on ion fluxes and the proteasomePyroptosis and host cell death responses during Salmonella infection.Streptolysin O promotes group A Streptococcus immune evasion by accelerated macrophage apoptosis.Thioredoxin 1 participates in the activity of the Salmonella enterica serovar Typhimurium pathogenicity island 2 type III secretion system.Immunity to salmonellosis.Understanding the mechanism of IL-1β secretionCaspase-1: is IL-1 just the tip of the ICEberg?The interplay between regulated necrosis and bacterial infection.Inflammasome Activation in Chronic Glomerular Diseases.Genetic requirements for salmonella-induced cytopathology in human monocyte-derived macrophages.Functions exerted by the virulence-associated type-three secretion systems during Salmonella enterica serovar Enteritidis invasion into and survival within chicken oviduct epithelial cells and macrophages.During infection of epithelial cells Salmonella enterica serovar Typhimurium undergoes a time-dependent transcriptional adaptation that results in simultaneous expression of three type 3 secretion systems.Cytokine response of porcine cell lines to Salmonella enterica serovar typhimurium and its hilA and ssrA mutants.NLRP3 activation induces ASC-dependent programmed necrotic cell death, which leads to neutrophilic inflammation.
P2860
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P2860
Salmonella pathogenicity island 2-dependent macrophage death is mediated in part by the host cysteine protease caspase-1.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh
2001年學術文章
@zh-hant
name
Salmonella pathogenicity islan ...... t cysteine protease caspase-1.
@en
type
label
Salmonella pathogenicity islan ...... t cysteine protease caspase-1.
@en
prefLabel
Salmonella pathogenicity islan ...... t cysteine protease caspase-1.
@en
P2093
P2860
P1476
Salmonella pathogenicity islan ...... t cysteine protease caspase-1.
@en
P2093
P2860
P304
P356
10.1046/J.1462-5822.2001.00162.X
P577
2001-12-01T00:00:00Z