Myocardial mechanical dysfunction following endotoxemia: role of changes in energy substrate metabolism.

about

Myocardial mechanical dysfunction following endotoxemia: role of changes in energy substrate metabolism.

Item

http://www.wikidata.org/entity/Q40773537

description

2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年学术文章

@wuu

2016年学术文章

@zh-cn

2016年学术文章

@zh-hans

2016年学术文章

@zh-my

2016年学术文章

@zh-sg

2016年學術文章

@yue

2016年學術文章

@zh

2016年學術文章

@zh-hant

name

Myocardial mechanical dysfunct ...... n energy substrate metabolism.

@en

type

Item

label

Myocardial mechanical dysfunct ...... n energy substrate metabolism.

@en

prefLabel

Myocardial mechanical dysfunct ...... n energy substrate metabolism.

@en

P1476

Myocardial mechanical dysfunct ...... in energy substrate metabolism

@en

P2093

Alexander S Clanachan

http://www.w3.org/2001/XMLSchema#string

Alireza Garjani

http://www.w3.org/2001/XMLSchema#string

Manoj Gandhi

http://www.w3.org/2001/XMLSchema#string

Waleed G T Masoud

http://www.w3.org/2001/XMLSchema#string

P2860

The mechanism of Intralipid®-mediated cardioprotection complex IV inhibition by the active metabolite, palmitoylcarnitine, generates reactive oxygen species and activates reperfusion injury salvage kinases

Impairment of glucose metabolism in hearts from rats treated with endotoxin

Depletion of myocardial glucose is observed during endotoxemic but not hemorrhagic shock in a porcine model.

Endotoxin pretreatment increases endogenous myocardial catalase activity and decreases ischemia-reperfusion injury of isolated rat hearts

Clinical utility of Doppler echocardiography and tissue Doppler imaging in the estimation of left ventricular filling pressures: A comparative simultaneous Doppler-catheterization study.

The Driving Force of the Na/Ca-Exchanger during Metabolic Inhibition.

Endotoxin-induced myocardial dysfunction in senescent rats

Diastolic dysfunction and mortality in severe sepsis and septic shock.

Myocardial substrate metabolism in the normal and failing heart.

Contribution of protons to post-ischemic Na(+) and Ca(2+) overload and left ventricular mechanical dysfunction.

P2888

s00395-016-0544-7

P304

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1007/S00395-016-0544-7

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

111

http://www.w3.org/2001/XMLSchema#string

P50

Hamid Soraya

P577

2016-02-29T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

296477015

http://www.w3.org/2001/XMLSchema#string

P698

26926341

http://www.w3.org/2001/XMLSchema#string