The molecular basis for paroxysmal nocturnal hemoglobinuria.
about
Solution structure of a functionally active fragment of decay-accelerating factorLeukemia arising out of paroxysmal nocturnal hemoglobinuria.The intrinsic complement regulator decay-accelerating factor modulates the biological response to vascular injuryImpaired growth and elevated fas receptor expression in PIGA(+) stem cells in primary paroxysmal nocturnal hemoglobinuriaGenotypic, immunophenotypic and clinical features of Thai patients with paroxysmal nocturnal haemoglobinuria.Tears contain the complement regulator CD59 as well as decay-accelerating factor (DAF).Chimaeric mice with disruption of the gene coding for phosphatidylinositol glycan class A (Pig-a) were defective in embryogenesis and spermatogenesis.Blockage of complement regulators in the conjunctiva and within the eye leads to massive inflammation and iritis.Protease-modified erythrocytes: CD55 and CD59 deficient PNH-like cells.
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P2860
The molecular basis for paroxysmal nocturnal hemoglobinuria.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
1993年學術文章
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1993年學術文章
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name
The molecular basis for paroxysmal nocturnal hemoglobinuria.
@en
type
label
The molecular basis for paroxysmal nocturnal hemoglobinuria.
@en
prefLabel
The molecular basis for paroxysmal nocturnal hemoglobinuria.
@en
P2093
P1433
P1476
The molecular basis for paroxysmal nocturnal hemoglobinuria.
@en
P2093
P304
P356
10.1046/J.1537-2995.1993.331094054626.X
P577
1993-10-01T00:00:00Z