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Transforming growth factor-beta: a clinical target for the treatment of diabetic nephropathyTGF-beta: a crucial component of the pathogenesis of diabetic nephropathyReversal of cardiac and renal fibrosis by pirfenidone and spironolactone in streptozotocin-diabetic ratsLaser capture microdissection/GeneChip analysis of gene expression in glomerular cells in diabetic db/db mice.Proinsulin C-peptide antagonizes the profibrotic effects of TGF-beta1 via up-regulation of retinoic acid and HGF-related signaling pathwaysAutocrine and paracrine mechanisms in the early stages of diabetic nephropathy.Soybeans ameliolate diabetic nephropathy in rats.Deletion of the receptor for advanced glycation end products reduces glomerulosclerosis and preserves renal function in the diabetic OVE26 mouse.Effect of metabolic syndrome on the response to arterial injuryRenal protective potential of antihypertensive drugs.Prevention of diabetic nephropathy in db/db mice with glycated albumin antagonists. A novel treatment strategy.Transforming growth factor β1 (TGF-β1) enhances expression of profibrotic genes through a novel signaling cascade and microRNAs in renal mesangial cellsRepression of let-7 by transforming growth factor-β1-induced Lin28 upregulates collagen expression in glomerular mesangial cells under diabetic conditions.G-CSF prevents progression of diabetic nephropathy in ratGrowth factor midkine is involved in the pathogenesis of diabetic nephropathy.Decorin interacts with connective tissue growth factor (CTGF)/CCN2 by LRR12 inhibiting its biological activitySimvastatin reverses podocyte injury but not mesangial expansion in early stage type 2 diabetes mellitusGlucose autoxidation induces functional damage to proteins via modification of critical arginine residuesCCN-2 is up-regulated by and mediates effects of matrix bound advanced glycated end-products in human renal mesangial cells.Renal accumulation of biglycan and lipid retention accelerates diabetic nephropathy.Long-term prevention of renal insufficiency, excess matrix gene expression, and glomerular mesangial matrix expansion by treatment with monoclonal antitransforming growth factor-beta antibody in db/db diabetic mice.17beta-Estradiol attenuates diabetic kidney disease by regulating extracellular matrix and transforming growth factor-beta protein expression and signalingKlotho gene delivery ameliorates renal hypertrophy and fibrosis in streptozotocin-induced diabetic rats by suppressing the Rho-associated coiled-coil kinase signaling pathway.MicroRNA-192 in diabetic kidney glomeruli and its function in TGF-beta-induced collagen expression via inhibition of E-box repressors.Glucose stimulation of transforming growth factor-beta bioactivity in mesangial cells is mediated by thrombospondin-1.Gelatinase B (MMP-9) is not essential in the normal kidney and does not influence progression of renal disease in a mouse model of Alport syndrome.The anti-inflammatory and antifibrotic effects of Coreopsis tinctoria Nutt on high-glucose-fat diet and streptozotocin-induced diabetic renal damage in rats.Advanced glycation end products up-regulate gene expression found in diabetic glomerular disease.Inhibiting microRNA-192 ameliorates renal fibrosis in diabetic nephropathy.MicroRNAs and the glomerulusA microRNA circuit mediates transforming growth factor-β1 autoregulation in renal glomerular mesangial cellsGene expression in diabetic nephropathy.Icariside II ameliorates diabetic nephropathy in streptozotocin-induced diabetic ratsMicroRNA circuits in transforming growth factor-β actions and diabetic nephropathy.Molecular basis for a link between complement and the vascular complications of diabetesBeneficial effects of thiazolidinediones on diabetic nephropathy in OLETF ratsTransforming growth factor-β-induced cross talk between p53 and a microRNA in the pathogenesis of diabetic nephropathyNOX enzymes and diabetic complications.Molecular medicine of microRNAs: structure, function and implications for diabetes.FOG2 protein down-regulation by transforming growth factor-β1-induced microRNA-200b/c leads to Akt kinase activation and glomerular mesangial hypertrophy related to diabetic nephropathy.
P2860
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P2860
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
1993年學術文章
@zh
1993年學術文章
@zh-hant
name
The extracellular matrix in diabetic nephropathy.
@en
type
label
The extracellular matrix in diabetic nephropathy.
@en
prefLabel
The extracellular matrix in diabetic nephropathy.
@en
P1476
The extracellular matrix in diabetic nephropathy.
@en
P2093
Ziyadeh FN
P304
P356
10.1016/S0272-6386(12)80440-9
P577
1993-11-01T00:00:00Z