Tumor necrosis factor as a mediator of shock, cachexia and inflammation.
about
TLR9 activation dampens the early inflammatory response to Paracoccidioides brasiliensis, impacting host survival.Improved survival and antagonistic effect of sodium fusidate on tumor necrosis factor alpha in a neonatal mouse model of endotoxin shockEndotoxin-induced lethality in neonatal mice is counteracted by interleukin-10 (IL-10) and exacerbated by anti-IL-10Synthesis and biological evaluation of novel propargylquinobenzothiazines and their derivatives as potential antiproliferative, anti-inflammatory, and anticancer agents.Inhibition of HIV-1 or bacterial activation of macrophages by products of HIV-1-resistant human cells.
P2860
Tumor necrosis factor as a mediator of shock, cachexia and inflammation.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
1993年學術文章
@zh
1993年學術文章
@zh-hant
name
Tumor necrosis factor as a mediator of shock, cachexia and inflammation.
@en
type
label
Tumor necrosis factor as a mediator of shock, cachexia and inflammation.
@en
prefLabel
Tumor necrosis factor as a mediator of shock, cachexia and inflammation.
@en
P356
P1433
P1476
Tumor necrosis factor as a mediator of shock, cachexia and inflammation.
@en
P2093
P304
P356
10.1159/000170104
P577
1993-01-01T00:00:00Z