Copper neurotoxicity is dependent on dopamine-mediated copper uptake and one-electron reduction of aminochrome in a rat substantia nigra neuronal cell line.
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P1343
Interactions of iron, dopamine and neuromelanin pathways in brain aging and Parkinson's diseaseExtracellular dopamine potentiates mn-induced oxidative stress, lifespan reduction, and dopaminergic neurodegeneration in a BLI-3-dependent manner in Caenorhabditis elegansTestosterone Propionate Exacerbates the Deficits of Nigrostriatal Dopaminergic System and Downregulates Nrf2 Expression in Reserpine-Treated Aged Male RatsAutophagy protects against aminochrome-induced cell death in substantia nigra-derived cell line.Comparative proteomic analysis of the molecular responses of mouse macrophages to titanium dioxide and copper oxide nanoparticles unravels some toxic mechanisms for copper oxide nanoparticles in macrophages.Overexpression of alpha-synuclein at non-toxic levels increases dopaminergic cell death induced by copper exposure via modulation of protein degradation pathways.Copper-induced structural conversion templates prion protein oligomerization and neurotoxicity.The association between Parkinson's disease and melanoma: a systematic review and meta-analysis.Nonsteroidal anti-inflammatory drugs in Parkinson's disease: possible involvement of quinone formation.Hypopituitarism Presenting as Adrenal Insufficiency and Hypothyroidism in a Patient with Wilson's Disease: a Case ReportYeast apurinic/apyrimidinic endonuclease Apn1 protects mammalian neuronal cell line from oxidative stress.Cuprous oxidase activity of CueO from Escherichia coli.Association of skin with the pathogenesis and treatment of neurodegenerative amyloidosisProtective and toxic roles of dopamine in Parkinson's disease.Metallomics insights into the programmed cell death induced by metal-based anticancer compounds.Copper increases the ability of 6-hydroxydopamine to generate oxidative stress and the ability of ascorbate and glutathione to potentiate this effect: potential implications in Parkinson's disease.Glutathione transferase mu 2 protects glioblastoma cells against aminochrome toxicity by preventing autophagy and lysosome dysfunction.One-electron reduction of 6-hydroxydopamine quinone is essential in 6-hydroxydopamine neurotoxicity.Overexpression of VMAT-2 and DT-diaphorase protects substantia nigra-derived cells against aminochrome neurotoxicity.Transition metal abnormalities in progressive dementias.Copper dopamine complex induces mitochondrial autophagy preceding caspase-independent apoptotic cell death.Linkage between catecholate siderophores and the multicopper oxidase CueO in Escherichia coli.Dopamine oxidation and autophagyCopper modulates the large dense core vesicle secretory pathway in PC12 cells.Protective effects of resveratrol on glutamate-induced damages in murine brain cultures.N-acetylcysteine attenuates copper overload-induced oxidative injury in brain of rat.Different mechanisms between copper and iron in catecholamines-mediated oxidative DNA damage and disruption of gene expression in vitro.Neurotoxicity of some MAO inhibitors in adult rat hypothalamic cell culture.Parkinson's Disease and Melanoma: Co-Occurrence and Mechanisms
P2860
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P2860
Copper neurotoxicity is dependent on dopamine-mediated copper uptake and one-electron reduction of aminochrome in a rat substantia nigra neuronal cell line.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
2001年论文
@zh
2001年论文
@zh-cn
name
Copper neurotoxicity is depend ...... ntia nigra neuronal cell line.
@en
type
label
Copper neurotoxicity is depend ...... ntia nigra neuronal cell line.
@en
prefLabel
Copper neurotoxicity is depend ...... ntia nigra neuronal cell line.
@en
P2093
P2860
P921
P1476
Copper neurotoxicity is depend ...... ntia nigra neuronal cell line.
@en
P2093
Bennett LB
Caviedes P
Caviedes R
Dagnino-Subiabre A
Marcelain K
P2860
P304
P356
10.1046/J.1471-4159.2001.00243.X
P407
P4510
P577
2001-04-01T00:00:00Z