A state of reversible compensated ventricular dysfunction precedes pathological remodelling in response to cardiomyocyte-specific activity of angiotensin II type-1 receptor in mice.

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Cardiomyocyte--specific expression of the nuclear matrix protein, CIZ1, stimulates production of mono-nucleated cells with an extended window of proliferation in the postnatal mouse heart.

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A state of reversible compensated ventricular dysfunction precedes pathological remodelling in response to cardiomyocyte-specific activity of angiotensin II type-1 receptor in mice.

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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A state of reversible compensa ...... in II type-1 receptor in mice.

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A state of reversible compensa ...... in II type-1 receptor in mice.

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A state of reversible compensa ...... in II type-1 receptor in mice.

@en

A state of reversible compensa ...... in II type-1 receptor in mice.

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A state of reversible compensa ...... in II type-1 receptor in mice.

@en

A state of reversible compensa ...... in II type-1 receptor in mice.

@en-gb

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2-s2.0-84939515262

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Disease Models & Mechanisms

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A state of reversible compensa ...... sin II type-1 receptor in mice

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Georgia A Frentzou

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Mark J Drinkhill

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Stephen G Ball

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P2860

Myosin heavy chain isoform expression in the failing and nonfailing human heart

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Inhibition of transforming growth factor-beta signaling induces left ventricular dilation and dysfunction in the pressure-overloaded heart

Relationship of interleukin-6 with regional and global left-ventricular function in asymptomatic individuals without clinical cardiovascular disease: insights from the Multi-Ethnic Study of Atherosclerosis.

How to explain the differences between renin angiotensin system modulators.

Regulation of peroxisome proliferator-activated receptor gamma coactivator 1 alpha (PGC-1 alpha ) and mitochondrial function by MEF2 and HDAC5

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783-794

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scholarly article

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10.1242/DMM.019174

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Neil A. Turner

Justin F Ainscough

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2015-06-18T00:00:00Z

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281392613

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89111