Molecular genetic studies of familial hypertrophic cardiomyopathy.
about
Phosphorylation of the regulatory light chain of myosin in striated muscle: methodological perspectivesAlterations at the intercalated disk associated with the absence of muscle LIM proteinFunctional consequences of caspase activation in cardiac myocytesGenetics of hypertrophic cardiomyopathy in eastern Finland: few founder mutations with benign or intermediary phenotypesCardiac myofilament regulation by protein phosphatase type 1alpha and CapZDiastolic dysfunction in familial hypertrophic cardiomyopathy transgenic model miceAngiotensin II blockade reverses myocardial fibrosis in a transgenic mouse model of human hypertrophic cardiomyopathyDilated cardiomyopathy in homozygous myosin-binding protein-C mutant miceA locus for autosomal dominant mitral valve prolapse on chromosome 11p15.4.The molecular genetic basis for hypertrophic cardiomyopathy.Molecular genetics and pathogenesis of hypertrophic cardiomyopathy.Control of cardiac myosin heavy chain gene expression.The genetic basis of cardiac function: dissection by zebrafish (Danio rerio) screens.Molecular medicine in the 21st century.Hypertrophic cardiomyopathy: from gene defect to clinical disease.Unequal allelic expression of wild-type and mutated β-myosin in familial hypertrophic cardiomyopathy.Structural and functional aspects of the myosin essential light chain in cardiac muscle contraction.Prevalence of aortic dilation in hypertrophic cardiomyopathy.Malignant familial hypertrophic cardiomyopathy D166V mutation in the ventricular myosin regulatory light chain causes profound effects in skinned and intact papillary muscle fibers from transgenic mice.Exon skipping in cardiac troponin T of turkeys with inherited dilated cardiomyopathy.From malignant mutations to malignant domains: the continuing search for prognostic significance in the mutant genes causing hypertrophic cardiomyopathy.Age-related changes in familial hypertrophic cardiomyopathy phenotype in transgenic mice and humans.Cardiac magnetic field map topology quantified by Kullback-Leibler entropy identifies patients with hypertrophic cardiomyopathy.A neuronal MAP kinase constrains growth of a C. elegans sensory dendrite throughout the life of the organism.
P2860
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P2860
Molecular genetic studies of familial hypertrophic cardiomyopathy.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh
1998年學術文章
@zh-hant
name
Molecular genetic studies of familial hypertrophic cardiomyopathy.
@en
type
label
Molecular genetic studies of familial hypertrophic cardiomyopathy.
@en
prefLabel
Molecular genetic studies of familial hypertrophic cardiomyopathy.
@en
P356
P1476
Molecular genetic studies of familial hypertrophic cardiomyopathy.
@en
P2093
Seidman CE
Seidman JG
P2888
P356
10.1007/S003950050196
P478
93 Suppl 3
P577
1998-01-01T00:00:00Z