An endoplasmic reticulum-specific stress-activated caspase (caspase-12) is implicated in the apoptosis of A549 epithelial cells by respiratory syncytial virus.
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Caspase-12 controls West Nile virus infection via the viral RNA receptor RIG-I.Japanese encephalitis virus infection initiates endoplasmic reticulum stress and an unfolded protein responseRespiratory syncytial virus infection sensitizes cells to apoptosis mediated by tumor necrosis factor-related apoptosis-inducing ligandActivation of cytokines and NF-kappa B in corneal epithelial cells infected by respiratory syncytial virus: potential relevance in ocular inflammation and respiratory infectionPhenotypic silencing of cytoplasmic genes using sequence-specific double-stranded short interfering RNA and its application in the reverse genetics of wild type negative-strand RNA virusesRespiratory Syncytial Virus and Cellular Stress Responses: Impact on Replication and PhysiopathologyHuman cytomegalovirus infection activates and regulates the unfolded protein response.Dengue virus serotype infection specifies the activation of the unfolded protein responseEndoplasmic Reticulum Stress Is a Determinant of Retrovirus-Induced Spongiform NeurodegenerationER Stress-Mediated Signaling: Action Potential and Ca(2+) as Key PlayersDifferential unfolded protein response during Chikungunya and Sindbis virus infection: CHIKV nsP4 suppresses eIF2α phosphorylationTumor necrosis factor induces apoptosis in hepatoma cells by increasing Ca(2+) release from the endoplasmic reticulum and suppressing Bcl-2 expressionResistance to vesicular stomatitis virus infection requires a functional cross talk between the eukaryotic translation initiation factor 2alpha kinases PERK and PKRMolecular components of a cell death pathway activated by endoplasmic reticulum stressTranscriptome characterization and gene expression of Epinephelus spp in endoplasmic reticulum stress-related pathway during betanodavirus infection in vitro.Confirmation by FRET in individual living cells of the absence of significant amyloid beta -mediated caspase 8 activationHeightened endoplasmic reticulum stress in the lungs of patients with chronic obstructive pulmonary disease: the role of Nrf2-regulated proteasomal activity.Anticancer oncolytic activity of respiratory syncytial virus.Vesicular stomatitis viruses expressing wild-type or mutant M proteins activate apoptosis through distinct pathways.Aquatic birnavirus-induced ER stress-mediated death signaling contribute to downregulation of Bcl-2 family proteins in salmon embryo cells.Hydrogen-rich saline alleviates early brain injury via reducing oxidative stress and brain edema following experimental subarachnoid hemorrhage in rabbits.A novel apoptotic pathway induced by the drs tumor suppressor gene.Viruses, endoplasmic reticulum stress, and interferon responses.Temporal activation of anti- and pro-apoptotic factors in human gingival fibroblasts infected with the periodontal pathogen, Porphyromonas gingivalis: potential role of bacterial proteases in host signalling.Caspase-12 and ER-stress-mediated apoptosis: the story so far.Uterine endoplasmic reticulum stress and its unfolded protein response may regulate caspase 3 activation in the pregnant mouse uterus.Endoplasmic reticulum stress and neurodegeneration in rats neonatally infected with borna disease virus.Role of apoptosis in sarcopenia.Mechanisms of neural cell death: implications for development of neuroprotective treatment strategiesRegulation of the expression and processing of caspase-12Roles of apoptosis in airway epithelia.Identification and functional characterization of tRNA-derived RNA fragments (tRFs) in respiratory syncytial virus infectionBioenergetics of murine lungs infected with respiratory syncytial virus.The group VIA calcium-independent phospholipase A2 participates in ER stress-induced INS-1 insulinoma cell apoptosis by promoting ceramide generation via hydrolysis of sphingomyelins by neutral sphingomyelinase.A link between endoplasmic reticulum stress-induced β-cell apoptosis and the group VIA Ca2+-independent phospholipase A2 (iPLA2β).Apoptosis of insulin-secreting cells induced by endoplasmic reticulum stress is amplified by overexpression of group VIA calcium-independent phospholipase A2 (iPLA2 beta) and suppressed by inhibition of iPLA2 beta.Human cytomegalovirus protein pUL38 induces ATF4 expression, inhibits persistent JNK phosphorylation, and suppresses endoplasmic reticulum stress-induced cell death.Enhancement of cisplatin [cis-diammine dichloroplatinum (II)] cytotoxicity by O6-benzylguanine involves endoplasmic reticulum stress.Conserved cysteine-rich domain of paramyxovirus simian virus 5 V protein plays an important role in blocking apoptosis.Antiviral RNAi: translating science towards therapeutic success.
P2860
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P2860
An endoplasmic reticulum-specific stress-activated caspase (caspase-12) is implicated in the apoptosis of A549 epithelial cells by respiratory syncytial virus.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh
2001年學術文章
@zh-hant
name
An endoplasmic reticulum-speci ...... y respiratory syncytial virus.
@en
type
label
An endoplasmic reticulum-speci ...... y respiratory syncytial virus.
@en
prefLabel
An endoplasmic reticulum-speci ...... y respiratory syncytial virus.
@en
P2860
P1476
An endoplasmic reticulum-speci ...... y respiratory syncytial virus.
@en
P2860
P304
P356
10.1002/1097-4644(20010301)80:3<441::AID-JCB170>3.0.CO;2-C
P577
2001-01-01T00:00:00Z