Interleukin-1beta induces apoptosis in GL15 glioblastoma-derived human cell line.
about
Calcium-mediated transductive systems and functionally active gap junctions in astrocyte-like GL15 cellsInterleukin-1β promotes hypoxia-induced apoptosis of glioblastoma cells by inhibiting hypoxia-inducible factor-1 mediated adrenomedullin productionM2 receptor activation inhibits cell cycle progression and survival in human glioblastoma cells.The antimicrobial peptide pardaxin exerts potent anti-tumor activity against canine perianal gland adenoma.The energy blockers bromopyruvate and lonidamine lead GL15 glioblastoma cells to death by different p53-dependent routes.Optimized mixture of As, Cd and Pb induce mitochondria-mediated apoptosis in C6-glioma via astroglial activation, inflammation and P38-MAPK.MAPK induces AQP1 expression in astrocytes following injury.Caspase activation in etoposide-treated fibroblasts is correlated to ERK phosphorylation and both events are blocked by polyamine depletion.
P2860
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P2860
Interleukin-1beta induces apoptosis in GL15 glioblastoma-derived human cell line.
description
2000 nî lūn-bûn
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name
Interleukin-1beta induces apoptosis in GL15 glioblastoma-derived human cell line.
@en
type
label
Interleukin-1beta induces apoptosis in GL15 glioblastoma-derived human cell line.
@en
prefLabel
Interleukin-1beta induces apoptosis in GL15 glioblastoma-derived human cell line.
@en
P2093
P2860
P921
P1476
Interleukin-1beta induces apoptosis in GL15 glioblastoma-derived human cell line
@en
P2093
E Castigli
G L Gianfranceschi
L Giovagnoli
V Bocchini
P2860
P304
P356
10.1152/AJPCELL.2000.279.6.C2043
P577
2000-12-01T00:00:00Z