Acute SGLT inhibition normalizes O2 tension in the renal cortex but causes hypoxia in the renal medulla in anaesthetized control and diabetic rats. - Wikidata - awgldk - TriplyDB

Acute SGLT inhibition normalizes O2 tension in the renal cortex but causes hypoxia in the renal medulla in anaesthetized control and diabetic rats.

Acute SGLT inhibition normalizes O2 tension in the renal cortex but causes hypoxia in the renal medulla in anaesthetized control and diabetic rats.

http://www.wikidata.org/entity/Q40871412

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Increased Hematocrit During Sodium-Glucose Cotransporter 2 Inhibitor Therapy Indicates Recovery of Tubulointerstitial Function in Diabetic Kidneys Dapagliflozin, SGLT2 Inhibitor, Attenuates Renal Ischemia-Reperfusion Injury Mechanisms of metabolic memory and renal hypoxia as a therapeutic target in diabetic kidney disease.Metabolic and hemodynamic effects of sodium-dependent glucose cotransporter 2 inhibitors on cardio-renal protection in the treatment of patients with type 2 diabetes mellitus Sodium glucose cotransporter 2 inhibition in the diabetic kidney: an update Predicted consequences of diabetes and SGLT inhibition on transport and oxygen consumption along a rat nephron.Recent advances in renal hypoxia: insights from bench experiments and computer simulations.Responses of renal hemodynamics and tubular functions to acute sodium-glucose cotransporter 2 inhibitor administration in non-diabetic anesthetized rats.Targeting renal glucose reabsorption to treat hyperglycaemia: the pleiotropic effects of SGLT2 inhibition.Hemodynamic Effects of Sodium-Glucose Cotransporter 2 Inhibitors.Increased Hematocrit During Sodium-Glucose Cotransporter-2 Inhibitor Therapy.Targeted mitochondrial therapy using MitoQ shows equivalent renoprotection to angiotensin converting enzyme inhibition but no combined synergy in diabetes.SGLT2 Inhibition in a Kidney with Reduced Nephron Number: Modeling and Analysis of Solute Transport and Metabolism.Sweet Success? SGLT2 Inhibitors and Diabetes.Can SGLT2 Inhibitors Cause Acute Renal Failure? Plausible Role for Altered Glomerular Hemodynamics and Medullary Hypoxia.Use of sodium-glucose cotransporter 2 inhibitors in older patients with type 2 diabetes mellitus.Cardiovascular benefits of SGLT2 inhibition in diabetes and chronic kidney diseases.Harnessing basic and clinic tools to evaluate SGLT2 inhibitor nephrotoxicity.New Diabetes Therapies and Diabetic Kidney Disease Progression: the Role of SGLT-2 Inhibitors.The Anticipated Renoprotective Effects of Sodium-glucose Cotransporter 2 Inhibitors.Experimental type II diabetes and related models of impaired glucose metabolism differentially regulate glucose transporters at the proximal tubule brush border membrane.Effects of the SGLT-2 inhibitor dapagliflozin on glomerular and tubular injury markers.Comprehensive renoprotective effects of ipragliflozin on early diabetic nephropathy in mice.Impact of Dapagliflozin Therapy on Renal Protection and Kidney Morphology in Patients With Uncontrolled Type 2 Diabetes Mellitus.Effect of canagliflozin on left ventricular diastolic function in patients with type 2 diabetes.

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P2860

Acute SGLT inhibition normalizes O2 tension in the renal cortex but causes hypoxia in the renal medulla in anaesthetized control and diabetic rats.

http://www.wikidata.org/entity/Q40871412

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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Acute SGLT inhibition normaliz ...... zed control and diabetic rats.

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Acute SGLT inhibition normaliz ...... zed control and diabetic rats.

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AJPRENAL.00689.2014

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American Journal of Physiology - Renal Physiology

P1476

Acute SGLT inhibition normaliz ...... ized control and diabetic rats

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Angelica Fasching

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Fredrik Palm

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Julie O'Neill

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Liselotte Pihl

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Stephanie Franzén

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P2860

Tubular sodium handling and tubuloglomerular feedback in experimental diabetes mellitus

Improved diabetic syndrome in C57BL/KsJ-db/db mice by oral administration of the Na(+)-glucose cotransporter inhibitor T-1095

Reactive oxygen species cause diabetes-induced decrease in renal oxygen tension.

Glomerular filtration and tubular reabsorption of albumin in preproteinuric and proteinuric diabetic rats.

Role of nitric oxide in renal medullary oxygenation. Studies in isolated and intact rat kidneys.

Acute knockdown of uncoupling protein-2 increases uncoupling via the adenine nucleotide transporter and decreases oxidative stress in diabetic kidneys

GLP-1 receptor activation and Epac2 link atrial natriuretic peptide secretion to control of blood pressure.

Diabetic nephropathy: diagnosis, prevention, and treatment.

SGLT2 mediates glucose reabsorption in the early proximal tubule.

Nitric oxide and angiotensin II. Glomerular and tubular interaction in the rat

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10.1152/AJPRENAL.00689.2014

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26041448

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