Cognate gene clusters govern invasion of host epithelial cells by Salmonella typhimurium and Shigella flexneri
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Selfish operons: horizontal transfer may drive the evolution of gene clustersIdentification of CsrC and characterization of its role in epithelial cell invasion in Salmonella enterica serovar TyphimuriumType III protein secretion systems in bacterial pathogens of animals and plantsCommon themes in microbial pathogenicity revisitedMolecular mechanisms of bacterial virulence: type III secretion and pathogenicity islandsBacterial type III secretion systems: specialized nanomachines for protein delivery into target cellsThe virulence plasmid of Yersinia, an antihost genomeVirulence and stress-related periplasmic protein (VisP) in bacterial/host associationsThe SseC translocon component in Salmonella enterica serovar Typhimurium is chaperoned by SscAYscO of Yersinia pestis is a mobile core component of the Yop secretion systemYscP of Yersinia pestis is a secreted component of the Yop secretion systemProtein secretion by enteropathogenic Escherichia coli is essential for transducing signals to epithelial cellsPhylogeny vs genome reshuffling: horizontal gene transferA naturally occurring single nucleotide polymorphism in the Salmonella SPI-2 type III effector srfH/sseI controls early extraintestinal disseminationGenome-wide screen of Salmonella genes expressed during infection in pigs, using in vivo expression technologyCoordinated regulation of virulence during systemic infection of Salmonella enterica serovar Typhimurium.Secretion of extracellular proteins by enterohemorrhagic Escherichia coli via a putative type III secretion system.The secreted Ipa complex of Shigella flexneri promotes entry into mammalian cellsMolecular basis of the interaction of Salmonella with the intestinal mucosa.The lpf fimbrial operon mediates adhesion of Salmonella typhimurium to murine Peyer's patches.QseC mediates Salmonella enterica serovar typhimurium virulence in vitro and in vivo.Identification of a novel Salmonella invasion locus homologous to Shigella ipgDEEnvironmental regulation of Salmonella typhi invasion-defective mutantsCell-contact-stimulated formation of filamentous appendages by Salmonella typhimurium does not depend on the type III secretion system encoded by Salmonella pathogenicity island 1.Inhibition of Salmonella typhimurium invasion by host cell expression of secreted bacterial invasion proteinsGene conservation and loss in the mutS-rpoS genomic region of pathogenic Escherichia coli.Analysis of the ArcA regulon in anaerobically grown Salmonella enterica sv. Typhimurium.Exposure to rumen protozoa leads to enhancement of pathogenicity of and invasion by multiple-antibiotic-resistant Salmonella enterica bearing SGI1.A periplasmic D-alanyl-D-alanine dipeptidase in the gram-negative bacterium Salmonella entericaHha is a negative modulator of transcription of hilA, the Salmonella enterica serovar Typhimurium invasion gene transcriptional activatorSalmonella typhimurium encodes a putative iron transport system within the centisome 63 pathogenicity island.Transcriptional organization and function of invasion genes within Salmonella enterica serovar Typhimurium pathogenicity island 1, including the prgH, prgI, prgJ, prgK, orgA, orgB, and orgC genes.Enteropathogenic Escherichia coli contains a putative type III secretion system necessary for the export of proteins involved in attaching and effacing lesion formationRelationship between evolutionary rate and cellular location among the Inv/Spa invasion proteins of Salmonella enterica.The players in a mutualistic symbiosis: insects, bacteria, viruses, and virulence genes.Organization and coordinated assembly of the type III secretion export apparatus.The flagellar regulator fliT represses Salmonella pathogenicity island 1 through flhDC and fliZ.A horizontally acquired transcription factor coordinates Salmonella adaptations to host microenvironments.Polyamines are required for virulence in Salmonella enterica serovar Typhimurium.Human genome-wide RNAi screen for host factors that modulate intracellular Salmonella growth.
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Cognate gene clusters govern invasion of host epithelial cells by Salmonella typhimurium and Shigella flexneri
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
1993年學術文章
@zh
1993年學術文章
@zh-hant
name
Cognate gene clusters govern i ...... himurium and Shigella flexneri
@en
type
label
Cognate gene clusters govern i ...... himurium and Shigella flexneri
@en
prefLabel
Cognate gene clusters govern i ...... himurium and Shigella flexneri
@en
P2860
P1433
P1476
Cognate gene clusters govern i ...... himurium and Shigella flexneri
@en
P2093
E A Groisman
P2860
P304
P356
10.1002/J.1460-2075.1993.TB06056.X
P407
P577
1993-10-01T00:00:00Z