Immunologic therapy for ARDS, septic shock, and multiple-organ failure.
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Protective effects of aerobic exercise on acute lung injury induced by LPS in miceRecombinant tissue factor pathway inhibitor reduces lipopolysaccharide-induced pulmonary vascular injury by inhibiting leukocyte activation.Role of Kupffer cells in acute hemorrhagic necrotizing pancreatitis-associated lung injury of rats.Intrapulmonary Hartmannella vermiformis: a potential niche for Legionella pneumophila replication in a murine model of legionellosis.The role of Legionella pneumophila-infected Hartmannella vermiformis as an infectious particle in a murine model of Legionnaire's diseaseRegulation of inflammatory responses by activated protein C: the molecular mechanism(s) and therapeutic implications.Lack of evidence for qualitative treatment by disease severity interactions in clinical studies of severe sepsis.Acute respiratory distress syndrome: a historical perspective.Agents to reduce cytokine storm.Targeting the host-pathogen interface for treatment of Staphylococcus aureus infection.Immunoglobulins in adult sepsis and septic shock.Induction of inflammatory cytokines in bovine alveolar macrophages following stimulation with Pasteurella haemolytica lipopolysaccharide.Noncardiac pulmonary edema, newer environmental aspects. An update.Cytokines in bronchoalveolar lavage fluid in patients with high altitude pulmonary oedema at moderate altitude in Japan.Why a second look might be worth it: immuno-modulatory therapies in the critically ill patient.The pneumococcus: host-organism interactions and their implications for immunotherapy and immunoprophylaxis.Corticotropin-releasing factor reduces lipopolysaccharide-induced pulmonary vascular leak.Influence of renal function on the pharmacokinetics of piperacillin/tazobactam in intensive care unit patients during continuous venovenous hemofiltration.In vivo nitric oxide detection in the septic rat brain by electron paramagnetic resonance.Capillary endothelial thrombomodulin expression and fibrin deposition in rats with continuous and bolus lipopolysaccharide administration.Exogenous surfactant decreases oxygenation in Escherichia coli endotoxin-treated neonatal piglets.
P2860
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P2860
Immunologic therapy for ARDS, septic shock, and multiple-organ failure.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
1993年學術文章
@zh
1993年學術文章
@zh-hant
name
Immunologic therapy for ARDS, septic shock, and multiple-organ failure.
@en
type
label
Immunologic therapy for ARDS, septic shock, and multiple-organ failure.
@en
prefLabel
Immunologic therapy for ARDS, septic shock, and multiple-organ failure.
@en
P356
P1433
P1476
Immunologic therapy for ARDS, septic shock, and multiple-organ failure.
@en
P2093
Dorinsky PM
St John RC
P304
P356
10.1378/CHEST.103.3.932
P407
P577
1993-03-01T00:00:00Z