Oncogene-induced senescence underlies the mutual exclusive nature of oncogenic KRAS and BRAF. - Wikidata - awgldk - TriplyDB

Oncogene-induced senescence underlies the mutual exclusive nature of oncogenic KRAS and BRAF.

Oncogene-induced senescence underlies the mutual exclusive nature of oncogenic KRAS and BRAF.

http://www.wikidata.org/entity/Q40885761

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BRAF Mutation in Colorectal Cancer: An Update The role of cell-free DNA in predicting colorectal cancer prognosis.Melanocytic nevi and melanoma: unraveling a complex relationship.Molecular Landscape of Acquired Resistance to Targeted Therapy Combinations in BRAF-Mutant Colorectal Cancer.What makes oncogenes mutually exclusive?Targeting Zfp148 activates p53 and reduces tumor initiation in the gut.The genomic landscape of malignant peripheral nerve sheath tumors: diverse drivers of Ras pathway activation.BRAF-Oncogene-Induced Senescence and the Role of Thyroid-Stimulating Hormone Signaling in the Progression of Papillary Thyroid Carcinoma.Identifying mutual exclusivity across cancer genomes: computational approaches to discover genetic interaction and reveal tumor vulnerability.p53 loss does not permit escape from BrafV600E-induced senescence in a mouse model of lung cancer.Acetylsalicylic Acid Governs the Effect of Sorafenib in RAS- Mutant Cancers.How Cancer Genomics Drives Cancer Biology: Does Synthetic Lethality Explain Mutually Exclusive Oncogenic Mutations?Novel clinicopathological and molecular characterization of metanephric adenoma: a study of 28 cases

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Oncogene-induced senescence underlies the mutual exclusive nature of oncogenic KRAS and BRAF.

http://www.wikidata.org/entity/Q40885761

description

2015 nî lūn-bûn

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2015年の論文

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年學術文章

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2015年學術文章

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2015年學術文章

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name

Oncogene-induced senescence un ...... re of oncogenic KRAS and BRAF.

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Oncogene-induced senescence un ...... re of oncogenic KRAS and BRAF.

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Oncogene-induced senescence un ...... re of oncogenic KRAS and BRAF.

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Oncogene-induced senescence un ...... ure of oncogenic KRAS and BRAF

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C Karlsson

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J Cisowski

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M O Bergo

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V I Sayin

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P2860

RAS oncogenes: weaving a tumorigenic web

The intermediate-activity (L597V)BRAF mutant acts as an epistatic modifier of oncogenic RAS by enhancing signaling through the RAF/MEK/ERK pathway

Ras oncogenes: split personalities

Conditional mouse lung cancer models using adenoviral or lentiviral delivery of Cre recombinase

Methods to detect biomarkers of cellular senescence: the senescence-associated beta-galactosidase assay.

Clinical pharmacogenomic testing of KRAS, BRAF and EGFR mutations by high resolution melting analysis and ultra-deep pyrosequencing

New driver mutations in non-small-cell lung cancer.

The regulation of INK4/ARF in cancer and aging.

Zfp148 deficiency causes lung maturation defects and lethality in newborn mice that are rescued by deletion of p53 or antioxidant treatment.

A new mouse model to explore the initiation, progression, and therapy of BRAFV600E-induced lung tumors.

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