Overexpression of catalase in the mitochondrial or cytosolic compartment increases sensitivity of HepG2 cells to tumor necrosis factor-alpha-induced apoptosis.
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Paradoxical Roles of Antioxidant Enzymes: Basic Mechanisms and Health ImplicationsA possible link between iron deficiency and gastrointestinal carcinogenesis.ALDOSE REDUCTASE: New Insights for an Old Enzyme.Is there more to aging than mitochondrial DNA and reactive oxygen species?Anti-inflammatory effect of targeted delivery of SOD to endothelium: mechanism, synergism with NO donors and protective effects in vitro and in vivo.Glutathione peroxidase-1 in health and disease: from molecular mechanisms to therapeutic opportunitiesEndothelial-derived oxidative stress drives myofibroblastic activation and calcification of the aortic valveAldose reductase mediates endotoxin-induced production of nitric oxide and cytotoxicity in murine macrophages.Antioxidant enzymes and apoptosis.Antioxidant dietary supplementation in mice exposed to proton radiation attenuates expression of programmed cell death-associated genes.Ethanol metabolism alters major histocompatibility complex class I-restricted antigen presentation in liver cellsAICAR inhibits oxygen consumption by intact skeletal muscle cells in culture.TNF-alpha mediates chemokine and cytokine expression and renal injury in cisplatin nephrotoxicity.Hypoxia triggers subcellular compartmental redox signaling in vascular smooth muscle cells.Relationship between oxidative stress levels and activation state on a hepatic stellate cell line.Riboflavin along with antibiotics balances reactive oxygen species and inflammatory cytokines and controls Staphylococcus aureus infection by boosting murine macrophage function and regulates inflammation.FOXO transcription factor activation by oxidative stress mediated by the small GTPase Ral and JNK.Resistance to tumor necrosis factor-alpha (TNF-alpha)-induced apoptosis in rat hepatoma cells expressing TNF-alpha is linked to low antioxidant enzyme expression.Histone acetylation and arachidonic acid cytotoxicity in HepG2 cells overexpressing CYP2E1.Mitochondrial reactive oxygen species regulate the temporal activation of nuclear factor kappaB to modulate tumour necrosis factor-induced apoptosis: evidence from mitochondria-targeted antioxidants.NADH oxidase activity of mitochondrial apoptosis-inducing factor.Mitochondrial dysfunction, persistent oxidative damage, and catalase inhibition in immune cells of naïve and treated Crohn's disease.Bile salt-phospholipid conjugate ursodeoxycholyl lysophosphatidylethanolamide as a hepatoprotective agent.Catalase down-regulation in cancer cells exposed to arsenic trioxide is involved in their increased sensitivity to a pro-oxidant treatment.Catalase and estradiol inhibit mitochondrial protein S-glutathionylation.
P2860
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P2860
Overexpression of catalase in the mitochondrial or cytosolic compartment increases sensitivity of HepG2 cells to tumor necrosis factor-alpha-induced apoptosis.
description
2000 nî lūn-bûn
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2000年学术文章
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name
Overexpression of catalase in ...... actor-alpha-induced apoptosis.
@en
type
label
Overexpression of catalase in ...... actor-alpha-induced apoptosis.
@en
prefLabel
Overexpression of catalase in ...... actor-alpha-induced apoptosis.
@en
P2860
P356
P1476
Overexpression of catalase in ...... actor-alpha-induced apoptosis.
@en
P2093
P2860
P304
19241-19249
P356
10.1074/JBC.M000438200
P407
P577
2000-06-01T00:00:00Z