PAI-1 gene expression is regionally induced in wounded epithelial cell monolayers and required for injury repair.
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Automated measurement of cell motility and proliferation.Dual role for plasminogen activator inhibitor type 1 as soluble and as matricellular regulator of epithelial alveolar cell wound healingSelective loss of TGFbeta Smad-dependent signalling prevents cell cycle arrest and promotes invasion in oesophageal adenocarcinoma cell linesA novel wounding device suitable for quantitative biochemical analysis of wound healing and regeneration of cultured epithelium.The effect of secretory factors of adipose-derived stem cells on human keratinocytes.The ternary complex factor Net regulates cell migration through inhibition of PAI-1 expressionPAI-1 Expression Is Required for HDACi-Induced Proliferative Arrest in ras-Transformed Renal Epithelial Cells.Hyperglycemia-suppressed expression of Serpine1 contributes to delayed epithelial wound healing in diabetic mouse corneas.The milieu of damaged alveolar epithelial type 2 cells stimulates alveolar wound repair by endogenous and exogenous progenitorsChemical Antagonists of Plasminogen Activator Inhibitor-1: Mechanisms of Action and Therapeutic Potential in Vascular Disease.PAI-1 and functional blockade of SNAI1 in breast cancer cell migration.Plasminogen activator inhibitor-1 does not contribute to the pulmonary pathology induced by acute exposure to ozone.uPA dependent and independent mechanisms of wound healing by C-phycocyaninLinking cell structure to gene regulation: signaling events and expression controls on the model genes PAI-1 and CTGF.Staufen-mediated mRNA decay.Antisense targeting of c-fos transcripts inhibits serum- and TGF-beta 1-stimulated PAI-1 gene expression and directed motility in renal epithelial cells.Stimulation of TLRs by LMW-HA induces self-defense mechanisms in vaginal epithelium.Bioinformatics analysis of potential essential genes that response to the high intraocular pressure on astrocyte due to glaucoma.Targeted Inhibition of PAI-1 Activity Impairs Epithelial Migration and Wound Closure Following Cutaneous Injury.Complex Regulation of the Pericellular Proteolytic Microenvironment during Tumor Progression and Wound Repair: Functional Interactions between the Serine Protease and Matrix Metalloproteinase Cascades.PAI-1: An Integrator of Cell Signaling and Migration.SERPINE1: A Molecular Switch in the Proliferation-Migration Dichotomy in Wound-"Activated" KeratinocytesA decisive function of transforming growth factor-β/Smad signaling in tissue morphogenesis and differentiation of human HaCaT keratinocytesDifferential requirement for MEK/ERK and SMAD signaling in PAI-1 and CTGF expression in response to microtubule disruption.
P2860
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P2860
PAI-1 gene expression is regionally induced in wounded epithelial cell monolayers and required for injury repair.
description
2000 nî lūn-bûn
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2000年の論文
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2000年学术文章
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2000年学术文章
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2000年学术文章
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2000年学术文章
@zh-my
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name
PAI-1 gene expression is regio ...... nd required for injury repair.
@en
type
label
PAI-1 gene expression is regio ...... nd required for injury repair.
@en
prefLabel
PAI-1 gene expression is regio ...... nd required for injury repair.
@en
P2093
P2860
P1476
PAI-1 gene expression is regio ...... nd required for injury repair.
@en
P2093
Higgins PJ
Providence KM
Staiano-Coico L
P2860
P304
P356
10.1002/(SICI)1097-4652(200002)182:2<269::AID-JCP16>3.0.CO;2-E
P577
2000-02-01T00:00:00Z