An optimized CD4 T-cell response can control productive and latent gammaherpesvirus infection
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CD4+ T-Cell Responses Are Required for Clearance of West Nile Virus from the Central Nervous SystemBlockade of virus infection by human CD4+ T cells via a cytokine relay networkEffective control of chronic gamma-herpesvirus infection by unconventional MHC Class Ia-independent CD8 T cells.Antibody-independent control of gamma-herpesvirus latency via B cell induction of anti-viral T cell responses.Murine gammaherpesvirus 68 infection is associated with lymphoproliferative disease and lymphoma in BALB beta2 microglobulin-deficient miceAntigen-loading compartments for major histocompatibility complex class II molecules continuously receive input from autophagosomes.Persistent gamma-herpesvirus infection induces a CD4 T cell response containing functionally distinct effector populations.Functional heterogeneity in the CD4+ T cell response to murine γ-herpesvirus 68Malignant catarrhal fever induced by Alcelaphine herpesvirus 1 is characterized by an expansion of activated CD3+CD8+CD4- T cells expressing a cytotoxic phenotype in both lymphoid and non-lymphoid tissuesInduction of TGF-beta 1, not regulatory T cells, impairs antiviral immunity in the lung following bone marrow transplant.Murine gammaherpesvirus 68 infection protects lupus-prone mice from the development of autoimmunity.Virus-specific CD4+ T cells: ready for direct attack.Autophagy and antigen presentation.A gammaherpesvirus-secreted activator of Vbeta4+ CD8+ T cells regulates chronic infection and immunopathology.Immunotherapy of human papilloma virus induced diseaseHerpesvirus interference with major histocompatibility complex class II-restricted T-cell activation.Expanding roles for CD4⁺ T cells in immunity to viruses.Race between retroviral spread and CD4+ T-cell response determines the outcome of acute Friend virus infection.CD4 CTL: living up to the challenge.Herpesviruses placating the unwilling host: manipulation of the MHC class II antigen presentation pathway.CD4 and CD8 T cells directly recognize murine gammaherpesvirus 68-immortalized cells and prevent tumor outgrowth.Specific mutation of a gammaherpesvirus-expressed antigen in response to CD8 T cell selection in vivo.Alpha/beta interferons regulate murine gammaherpesvirus latent gene expression and reactivation from latency.Pathology of immunodeficient mice with naturally occurring murine norovirus infection.Critical role of CD4 T cells in an antibody-independent mechanism of vaccination against gammaherpesvirus latency.CD4 T cells mediate killing during persistent gammaherpesvirus 68 infection.Conserved herpesviral kinase promotes viral persistence by inhibiting the IRF-3-mediated type I interferon response.Adoptive transfer of cytomegalovirus-specific effector CD4+ T cells provides antiviral protection from murine CMV infection.Advances in the Characterization of the T-Cell Response to Human Herpesvirus-6.
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P2860
An optimized CD4 T-cell response can control productive and latent gammaherpesvirus infection
description
2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
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name
An optimized CD4 T-cell respon ...... ent gammaherpesvirus infection
@en
type
label
An optimized CD4 T-cell respon ...... ent gammaherpesvirus infection
@en
prefLabel
An optimized CD4 T-cell respon ...... ent gammaherpesvirus infection
@en
P2093
P2860
P1433
P1476
An optimized CD4 T-cell respon ...... ent gammaherpesvirus infection
@en
P2093
Douglas C Braaten
Rebecca L Sparks-Thissen
Samuel H Speck
Scott Kreher
P2860
P304
P356
10.1128/JVI.78.13.6827-6835.2004
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P577
2004-07-01T00:00:00Z