Enhanced sensitivity of pituitary beta-endorphin to ethanol in subjects at high risk of alcoholism.
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An evaluation of mu-opioid receptor (OPRM1) as a predictor of naltrexone response in the treatment of alcohol dependence: results from the Combined Pharmacotherapies and Behavioral Interventions for Alcohol Dependence (COMBINE) studySubjective responses to alcohol prime event-specific alcohol consumption and predict blackouts and hangoverPharmacological evidence for a motivational role of kappa-opioid systems in ethanol dependenceInfluence of the endogenous opioid system on high alcohol consumption and genetic predisposition to alcoholismLong-term opioid blockade and hedonic response: preliminary data from two open-label extension studies with extended-release naltrexonePreliminary data on the association among the serotonin transporter polymorphism, subjective alcohol experiences, and drinking behaviorWorld Federation of Societies of Biological Psychiatry (WFSBP) Guidelines for Biological Treatment of Substance Use and Related Disorders, Part 1: Alcoholism.Potent inhibition of alcohol self-administration in alcohol-preferring rats by a κ-opioid receptor antagonistSynthesis and pharmacological evaluation of 6-naltrexamine analogs for alcohol cessation.Involvement of the beta-endorphin neurons of the hypothalamic arcuate nucleus in ethanol-induced place preference conditioning in miceSeverity of alcoholism in Indian males: Correlation with age of onset and family history of alcoholism.Opioid antagonists in the treatment of alcohol dependence.A functional polymorphism of the mu-opioid receptor gene (OPRM1) influences cue-induced craving for alcohol in male heavy drinkers.Selection for high alcohol preference drinking in mice results in heightened sensitivity and rapid development of acute functional tolerance to alcohol's ataxic effects.Beta-endorphin mediates behavioral despair and the effect of ethanol on the tail suspension test in miceHIV, alcohol dependence, and the criminal justice system: a review and call for evidence-based treatment for released prisoners.Differential effects of naltrexone on cardiac, subjective and behavioural reactions to acute ethanol intoxicationAlcoholism: the dissection for endophenotypes.Neuropeptides: implications for alcoholism.Targeting dynorphin/kappa opioid receptor systems to treat alcohol abuse and dependence.Guidelines for biological treatment of substance use and related disorders, part 1: Alcoholism, first revision.Alcohol impairment of saccadic and smooth pursuit eye movements: impact of risk factors for alcohol dependenceAlcohol-induced plasticity in the dynorphin/kappa-opioid receptor system.Neurobiology of addiction: treatment and public policy ramifications.Mu opioid receptor (OPRM1) as a predictor of treatment outcome in opiate-dependent individuals of Arab descent.Targeting treatments for alcohol dependence: the pharmacogenetics of naltrexone.Conceptualizing withdrawal-induced escalation of alcohol self-administration as a learned, plasticity-dependent process.Effects of naltrexone on cortisol levels in heavy drinkers.Delta and kappa opioid receptor polymorphisms influence the effects of naltrexone on subjective responses to alcohol.Polymorphisms in the mu-opioid receptor gene (OPRM1) and the implications for alcohol dependence in humans.Pharmacogenetic treatments for drug addiction: alcohol and opiates.Penn/VA center for studies of addiction.Sweet liking phenotype, alcohol craving and response to naltrexone treatment in alcohol dependence.Naltrexone alone and with sertraline for the treatment of alcohol dependence in Alaska natives and non-natives residing in rural settings: a randomized controlled trialAppetitive motivational experience during adolescence results in enhanced alcohol consumption during adulthood.κ-opioid receptors are implicated in the increased potency of intra-accumbens nalmefene in ethanol-dependent rats.The obesity epidemic and food addiction: clinical similarities to drug dependence.Influence of the OPRM1 A118G polymorphism on alcohol-induced euphoria, risk for alcoholism and the clinical efficacy of naltrexone.Mechanical Stimulation of the HT7 Acupuncture Point to Reduce Ethanol Self-Administration in Rats.Alcohol, stress, and glucocorticoids: From risk to dependence and relapse in alcohol use disorders.
P2860
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P2860
Enhanced sensitivity of pituitary beta-endorphin to ethanol in subjects at high risk of alcoholism.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Enhanced sensitivity of pituit ...... ts at high risk of alcoholism.
@en
type
label
Enhanced sensitivity of pituit ...... ts at high risk of alcoholism.
@en
prefLabel
Enhanced sensitivity of pituit ...... ts at high risk of alcoholism.
@en
P2093
P1433
P1476
Enhanced sensitivity of pituit ...... ts at high risk of alcoholism.
@en
P2093
Gianoulakis C
Krishnan B
Thavundayil J
P304
P356
10.1001/ARCHPSYC.1996.01830030072011
P407
P577
1996-03-01T00:00:00Z