ETS-1 converts endothelial cells to the angiogenic phenotype by inducing the expression of matrix metalloproteinases and integrin beta3.
about
The biology of the Ets1 proto-oncogeneOverexpression of the p53-inducible brain-specific angiogenesis inhibitor 1 suppresses efficiently tumour angiogenesisPGC-1alpha inhibits oleic acid induced proliferation and migration of rat vascular smooth muscle cells.Ets-1 regulates energy metabolism in cancer cells.Biophysiochemical properties of endothelial cells cultured on bio-inspired collagen films.Ets-1 is a critical regulator of Ang II-mediated vascular inflammation and remodelingMatrix metalloproteinase control of capillary morphogenesis.The transcription factor ETS-1 mediates proinflammatory responses and neointima formation in carotid artery endoluminal vascular injury.Ets-1 global gene expression profile reveals associations with metabolism and oxidative stress in ovarian and breast cancersmiR-200b targets Ets-1 and is down-regulated by hypoxia to induce angiogenic response of endothelial cells.Hypoxia-inducible factors modulate the stemness and malignancy of colon cancer cells by playing opposite roles in canonical Wnt signaling.Transcriptional silencing of ETS-1 abrogates epithelial-mesenchymal transition resulting in reduced motility of pancreatic cancer cellsAbnormal expression of FLI1 protein is an adverse prognostic factor in acute myeloid leukemiaEts1 transcription factor mediates gastrin-releasing peptide-induced IL-8 regulation in neuroblastoma cells.Blockade of Ets-1 attenuates epidermal growth factor-dependent collagen loss in human carotid plaque smooth muscle cells.Ets-1 as an early response gene against hypoxia-induced apoptosis in pancreatic β-cellsETS transcription factors in embryonic vascular development.Noninvasive delivery of gene targeting probes to live brains for transcription MRIEts-1 p51 and p42 isoforms differentially modulate Stromelysin-1 promoter according to induced DNA bend orientation.Matrix metalloproteinase-9: Many shades of function in cardiovascular disease.Poly-N-acetyl glucosamine nanofibers regulate endothelial cell movement and angiogenesis: dependency on integrin activation of Ets1.Passing the baton: the HIF switch.Transcription factor Ets-1 mediates ischemia- and vascular endothelial growth factor-dependent retinal neovascularization.Silencing of ETS1 reverses adriamycin resistance in MCF-7/ADR cells via downregulation of MDR1.Predicting cell-type-specific gene expression from regions of open chromatin.c-ETS1 facilitates G1/S-phase transition by up-regulating cyclin E and CDK2 genes and cooperates with hepatitis B virus X protein for their deregulationThe hypoxia-associated factor switches cells from HIF-1α- to HIF-2α-dependent signaling promoting stem cell characteristics, aggressive tumor growth and invasion.Ets2 and protein kinase C epsilon are important regulators of parathyroid hormone-related protein expression in MCF-7 breast cancer cells.Roles of extracellular signal-regulated kinase 1/2 and p38 mitogen-activated protein kinase in the signal transduction of basic fibroblast growth factor in endothelial cells during angiogenesis.VEGF amplifies transcription through ETS1 acetylation to enable angiogenesis.Camptothecin poly[n-(2-hydroxypropyl) methacrylamide] copolymers in antitopoisomerase-I tumor therapy: intratumor release and antitumor efficacy.
P2860
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P2860
ETS-1 converts endothelial cells to the angiogenic phenotype by inducing the expression of matrix metalloproteinases and integrin beta3.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
ETS-1 converts endothelial cel ...... roteinases and integrin beta3.
@en
type
label
ETS-1 converts endothelial cel ...... roteinases and integrin beta3.
@en
prefLabel
ETS-1 converts endothelial cel ...... roteinases and integrin beta3.
@en
P2093
P1476
ETS-1 converts endothelial cel ...... roteinases and integrin beta3.
@en
P2093
P304
P356
10.1002/(SICI)1097-4652(199902)178:2<121::AID-JCP1>3.3.CO;2-6
P577
1999-02-01T00:00:00Z