Celastrol inhibits migration and invasion through blocking the NF-κB pathway in ovarian cancer cells.

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Lipid metabolism and carcinogenesis, cancer development.The coordinated effects of Apatinib and Tripterine on the proliferation, invasiveness and apoptosis of human hepatoma Hep3B cells.

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Celastrol inhibits migration and invasion through blocking the NF-κB pathway in ovarian cancer cells.

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2017 nî lūn-bûn

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2017年の論文

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Celastrol inhibits migration a ...... thway in ovarian cancer cells.

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Celastrol inhibits migration a ...... thway in ovarian cancer cells.

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Celastrol inhibits migration a ...... thway in ovarian cancer cells.

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Xiulan Du

http://www.w3.org/2001/XMLSchema#string

Zhenyuan Zhai

http://www.w3.org/2001/XMLSchema#string

Zhongye Wang

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Rethinking ovarian cancer II: reducing mortality from high-grade serous ovarian cancer

Celastrol suppresses tumor cell growth through targeting an AR-ERG-NF-κB pathway in TMPRSS2/ERG fusion gene expressing prostate cancer

NF-kappa B modulation is involved in celastrol induced human multiple myeloma cell apoptosis

Tumour-cell invasion and migration: diversity and escape mechanisms

NF-kappaB: linking inflammation and immunity to cancer development and progression

NF-kappaB in cancer: from innocent bystander to major culprit

Natural proteasome inhibitor celastrol suppresses androgen-independent prostate cancer progression by modulating apoptotic proteins and NF-kappaB

TNFalpha up-regulates SLUG via the NF-kappaB/HIF1alpha axis, which imparts breast cancer cells with a stem cell-like phenotype.

Bmi-1 promotes the aggressiveness of glioma via activating the NF-kappaB/MMP-9 signaling pathway.

Celastrol suppresses invasion of colon and pancreatic cancer cells through the downregulation of expression of CXCR4 chemokine receptor.

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scholarly article

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10.3892/ETM.2017.4568

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2017-06-08T00:00:00Z

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28673005

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ovarian cancer

NF-κB

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5488742

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Celastrol inhibits migration and invasion through blocking the NF-κB pathway in ovarian cancer cells.

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P2860

P2860

Celastrol inhibits migration and invasion through blocking the NF-κB pathway in ovarian cancer cells.

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