Presynaptic N-Methyl-d-aspartate (NMDA) Receptor Activity Is Increased Through Protein Kinase C in Paclitaxel-induced Neuropathic Pain
about
From Mechanism to Cure: Renewing the Goal to Eliminate the Disease of Pain.Presynaptic mGluR5 receptor controls glutamatergic input through protein kinase C-NMDA receptors in paclitaxel-induced neuropathic pain.Bortezomib induces neuropathic pain through protein kinase C-mediated activation of presynaptic NMDA receptors in the spinal cord.The α2δ-1-NMDA Receptor Complex Is Critically Involved in Neuropathic Pain Development and Gabapentin Therapeutic Actions.
P2860
Presynaptic N-Methyl-d-aspartate (NMDA) Receptor Activity Is Increased Through Protein Kinase C in Paclitaxel-induced Neuropathic Pain
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Presynaptic N-Methyl-d-asparta ...... taxel-induced Neuropathic Pain
@en
type
label
Presynaptic N-Methyl-d-asparta ...... taxel-induced Neuropathic Pain
@en
prefLabel
Presynaptic N-Methyl-d-asparta ...... taxel-induced Neuropathic Pain
@en
P2093
P2860
P356
P1476
Presynaptic N-Methyl-d-asparta ...... taxel-induced Neuropathic Pain
@en
P2093
Hui-Lin Pan
Jing-Dun Xie
Shao-Rui Chen
Wei-An Zeng
P2860
P304
19364-19373
P356
10.1074/JBC.M116.732347
P407
P577
2016-07-25T00:00:00Z