Intrathecal heat shock protein 60 mediates neurodegeneration and demyelination in the CNS through a TLR4- and MyD88-dependent pathway.
about
Immune Surveillance of the CNS following Infection and InjuryMicroglia activation triggers oligodendrocyte precursor cells apoptosis via HSP60.HSP60 plays a regulatory role in IL-1β-induced microglial inflammation via TLR4-p38 MAPK axis.Oxymatrine inhibits microglia activation via HSP60-TLR4 signaling.Sterile Neuroinflammation and Strategies for Therapeutic InterventionNeuroprotective effect of heat shock protein 60 on matrine-suppressed microglial activation.CD40 Negatively Regulates ATP-TLR4-Activated Inflammasome in Microglia.Mitochondrial Chaperones in the Brain: Safeguarding Brain Health and Metabolism?
P2860
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P2860
Intrathecal heat shock protein 60 mediates neurodegeneration and demyelination in the CNS through a TLR4- and MyD88-dependent pathway.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Intrathecal heat shock protein ...... - and MyD88-dependent pathway.
@en
type
label
Intrathecal heat shock protein ...... - and MyD88-dependent pathway.
@en
prefLabel
Intrathecal heat shock protein ...... - and MyD88-dependent pathway.
@en
P2093
P2860
P50
P1476
Intrathecal heat shock protein ...... - and MyD88-dependent pathway.
@en
P2093
Christina Krüger
Eckart Schott
Karen Rosenberger
Katja Derkow
Paul Dembny
Seija Lehnardt
P2860
P2888
P356
10.1186/S13024-015-0003-1
P577
2015-02-26T00:00:00Z
P5875
P6179
1000153175