Sodium channel protein expression enhances the invasiveness of rat and human prostate cancer cells.
about
STEAP: a prostate-specific cell-surface antigen highly expressed in human prostate tumorsEpithelial Na, K-ATPase expression is down-regulated in canine prostate cancer; a possible consequence of metabolic transformation in the process of prostate malignancyVoltage-gated sodium channels and cancer: is excitability their primary role?Increased expression of fibroblast growth factor 13 in prostate cancer is associated with shortened time to biochemical recurrence after radical prostatectomy.Expression of skeletal muscle-type voltage-gated Na+ channel in rat and human prostate cancer cell linesCancer: fundamentals behind pH targeting and the double-edged approachAsymmetric synthesis and evaluation of a hydroxyphenylamide voltage-gated sodium channel blocker in human prostate cancer xenograftssiRNA knockdown of ribosomal protein gene RPL19 abrogates the aggressive phenotype of human prostate cancer.The cellular and molecular basis of prostate cancer.Voltage-gated sodium channels were differentially expressed in human normal prostate, benign prostatic hyperplasia and prostate cancer cells.The neonatal splice variant of Nav1.5 potentiates in vitro invasive behaviour of MDA-MB-231 human breast cancer cells.Increased expression of anterior gradient-2 is significantly associated with poor survival of prostate cancer patients.PRKC-ζ Expression Promotes the Aggressive Phenotype of Human Prostate Cancer Cells and Is a Novel Target for Therapeutic Intervention.A role for STEAP2 in prostate cancer progression.Epidermal growth factor, neurotrophins and the metastatic cascade in prostate cancer.An emerging role for voltage-gated Na+ channels in cellular migration: regulation of central nervous system development and potentiation of invasive cancersEpidermal growth factor potentiates in vitro metastatic behaviour of human prostate cancer PC-3M cells: involvement of voltage-gated sodium channel.Voltage-gated sodium channels and metastatic disease.Sigma receptors [σRs]: biology in normal and diseased statesIon channels as targets for cancer therapyRoles of ion transport in control of cell motility.Regulation of voltage-gated sodium channel expression in cancer: hormones, growth factors and auto-regulation.Voltage-gated sodium channel as a target for metastatic risk reduction with re-purposed drugs.Six transmembrane epithelial antigen of the prostate 1 is down-regulated by sex hormones in prostate cells.Sodium ion channel mutations in glioblastoma patients correlate with shorter survival.Eicosapentaenoic acid inhibits voltage-gated sodium channels and invasiveness in prostate cancer cells.Nerve growth factor enhances voltage-gated Na+ channel activity and Transwell migration in Mat-LyLu rat prostate cancer cell line.Notch signalling and voltage-gated Na+ channel activity in human prostate cancer cells: independent modulation of in vitro motility.Activity-dependent regulation of voltage-gated Na+ channel expression in Mat-LyLu rat prostate cancer cell line.Contact stimulation of prostate cancer cell migration: the role of gap junctional coupling and migration stimulated by heterotypic cell-to-cell contacts in determination of the metastatic phenotype of Dunning rat prostate cancer cells.Expression of Na+-dependent citrate transport in a strongly metastatic human prostate cancer PC-3M cell line: regulation by voltage-gated Na+ channel activity.Effects of voltage-gated ion channel modulators on rat prostatic cancer cell proliferation: comparison of strongly and weakly metastatic cell lines.Effects of Hedera helix L. extracts on rat prostate cancer cell proliferation and motility.Absence of SPARC leads to impaired lens circulation.A novel cutaneous Fatty Acid-binding protein-related signaling pathway leading to malignant progression in prostate cancer cells.Expression of Voltage-Gated Sodium Channel Nav1.8 in Human Prostate Cancer is Associated with High Histological Grade.Expression profiles of voltage-gated Na(+) channel alpha-subunit genes in rat and human prostate cancer cell lines.Epidermal growth factor upregulates motility of Mat-LyLu rat prostate cancer cells partially via voltage-gated Na+ channel activity.Protein kinase C isoenzyme patterns characteristically modulated in early prostate cancerPredominant expression of Kv1.3 voltage-gated K+ channel subunit in rat prostate cancer cell lines: electrophysiological, pharmacological and molecular characterisation.
P2860
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P2860
Sodium channel protein expression enhances the invasiveness of rat and human prostate cancer cells.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh
1998年學術文章
@zh-hant
name
Sodium channel protein express ...... d human prostate cancer cells.
@en
type
label
Sodium channel protein express ...... d human prostate cancer cells.
@en
prefLabel
Sodium channel protein express ...... d human prostate cancer cells.
@en
P2093
P2860
P1433
P1476
Sodium channel protein express ...... d human prostate cancer cells.
@en
P2093
Djamgoz MB
Shortland AP
P2860
P356
10.1016/S0014-5793(98)00050-7
P407
P577
1998-02-01T00:00:00Z