SDF1-CXCR4 Signaling Maintains Central Post-Stroke Pain through Mediation of Glial-Neuronal Interactions

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Sinomenine attenuates cancer-induced bone pain via suppressing microglial JAK2/STAT3 and neuronal CAMKII/CREB cascades in rat models

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SDF1-CXCR4 Signaling Maintains Central Post-Stroke Pain through Mediation of Glial-Neuronal Interactions

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2017 nî lūn-bûn

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SDF1-CXCR4 Signaling Maintains ...... of Glial-Neuronal Interactions

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SDF1-CXCR4 Signaling Maintains ...... of Glial-Neuronal Interactions

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SDF1-CXCR4 Signaling Maintains ...... of Glial-Neuronal Interactions

@en

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SDF1-CXCR4 Signaling Maintains ...... of Glial-Neuronal Interactions

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Chun-Li Li

http://www.w3.org/2001/XMLSchema#string

Fan Yang

http://www.w3.org/2001/XMLSchema#string

Fei Yang

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Jiang-Lin Wang

http://www.w3.org/2001/XMLSchema#string

Jun Chen

http://www.w3.org/2001/XMLSchema#string

Na Wei

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Su-Min Guan

http://www.w3.org/2001/XMLSchema#string

Wei Sun

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Wen-Jun Luo

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Xiao-Liang Wang

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CXCR4 signaling mediates morphine-induced tactile hyperalgesia.

Global and regional burden of stroke during 1990-2010: findings from the Global Burden of Disease Study 2010

Modulation of thalamic nociceptive processing after spinal cord injury through remote activation of thalamic microglia by cysteine cysteine chemokine ligand 21

Chemokines and glycoprotein120 produce pain hypersensitivity by directly exciting primary nociceptive neurons

Delayed functional expression of neuronal chemokine receptors following focal nerve demyelination in the rat: a mechanism for the development of chronic sensitization of peripheral nociceptors

Modulation of cocaine-induced activity by intracerebral administration of CXCL12

Neuronal and glial alterations due to focal cortical hypoxia induced by direct cobalt chloride (CoCl2) brain injection

CXCR4 chemokine receptor signaling mediates pain hypersensitivity in association with antiretroviral toxic neuropathy.

Increased chemokine signaling in a model of HIV1-associated peripheral neuropathy.

Down-Regulation of CXCL12/CXCR4 Expression Alleviates Ischemia-Reperfusion-Induced Inflammatory Pain via Inhibiting Glial TLR4 Activation in the Spinal Cord.

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226

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scholarly article

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10.3389/FNMOL.2017.00226

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2017-07-21T00:00:00Z

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28785202

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5519565

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SDF1-CXCR4 Signaling Maintains Central Post-Stroke Pain through Mediation of Glial-Neuronal Interactions

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SDF1-CXCR4 Signaling Maintains Central Post-Stroke Pain through Mediation of Glial-Neuronal Interactions

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