Different roles for cytotoxic T cells in the control of infections with cytopathic versus noncytopathic viruses.
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Natural pathogens of laboratory mice, rats, and rabbits and their effects on research.Direct cleavage of the human DNA fragmentation factor-45 by granzyme B induces caspase-activated DNase release and DNA fragmentationHuman perforin mutations and susceptibility to multiple primary cancersEvaluating the cytotoxicity of innate immune effector cells using the GrB ELISPOT assayThe Granzyme B ELISPOT assay: an alternative to the 51Cr-release assay for monitoring cell-mediated cytotoxicityInduction of protective immunity against Japanese encephalitis in mice by immunization with a plasmid encoding Japanese encephalitis virus premembrane and envelope genes.Alphavirus-specific cytotoxic T lymphocytes recognize a cross-reactive epitope from the capsid protein and can eliminate virus from persistently infected macrophagesManaging Occupational Risks for Hepatitis C Transmission in the Health Care SettingVaccines for Venezuelan equine encephalitisContrasting effects of CCR5 and CCR2 deficiency in the pulmonary inflammatory response to influenza A virusCa2+ signaling modulates cytolytic T lymphocyte effector functions.NK cells and apoptosis.Protection from lethal coxsackievirus-induced pancreatitis by expression of gamma interferon.Towards an understanding of the herpes simplex virus type 1 latency-reactivation cycleThe relative role of lymphocyte granule exocytosis versus death receptor-mediated cytotoxicity in viral pathophysiologyTheiler's virus infection of perforin-deficient mice.Protection against lethal encephalomyocarditis virus infection in the absence of serum-neutralizing antibodies.Apoptosis and regeneration of hepatocytes during recovery from transient hepadnavirus infectionsGamma interferon is not required for mucosal cytotoxic T-lymphocyte responses or heterosubtypic immunity to influenza A virus infection in mice.The virus-specific and allospecific cytotoxic T-lymphocyte response to lymphocytic choriomeningitis virus is modified in a subpopulation of CD8(+) T cells coexpressing the inhibitory major histocompatibility complex class I receptor Ly49G2Epidermal powder immunization induces both cytotoxic T-lymphocyte and antibody responses to protein antigens of influenza and hepatitis B viruses.The role of antigen-independent persistence of memory cytotoxic T lymphocytes.Kinetic analysis of the specific host response to a murine gammaherpesvirusClearance of herpes simplex virus type 2 by CD8+ T cells requires gamma interferon and either perforin- or Fas-mediated cytolytic mechanismsHost factors influencing viral persistence.High-level primary CD8(+) T-cell response to human immunodeficiency virus type 1 gag and env generated by vaccination with recombinant vesicular stomatitis viruses.Control or persistence of hepatitis B virus: the critical role of initial host-virus interactions.Perforin-mediated target-cell death and immune homeostasis.Impaired CD8(+) T cell immunity after allogeneic bone marrow transplantation leads to persistent and severe respiratory viral infection.Granzymes and perforin in solid organ transplant rejection.Perforin is a critical physiologic regulator of T-cell activation.Control of mucosal virus infection by influenza nucleoprotein-specific CD8+ cytotoxic T lymphocytesPhenotypic and functional analyses of NK and NKT-like populations during the early stages of chikungunya infectionHerpes simplex virus 1 induces and blocks apoptosis at multiple steps during infection and protects cells from exogenous inducers in a cell-type-dependent mannerT cell memory in the lung airways.CXCL9 and CXCL10 expression are critical for control of genital herpes simplex virus type 2 infection through mobilization of HSV-specific CTL and NK cells to the nervous systemAddressing the mysteries of perforin function.Structural features of peptide analogs of human histocompatibility leukocyte antigen class I epitopes that are more potent and immunogenic than wild-type peptide.Contribution of virus-specific CD8+ cytotoxic T cells to virus clearance or pathologic manifestations of influenza virus infection in a T cell receptor transgenic mouse model.Type I interferons act directly on CD8 T cells to allow clonal expansion and memory formation in response to viral infection.
P2860
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P2860
Different roles for cytotoxic T cells in the control of infections with cytopathic versus noncytopathic viruses.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Different roles for cytotoxic ...... versus noncytopathic viruses.
@en
type
label
Different roles for cytotoxic ...... versus noncytopathic viruses.
@en
prefLabel
Different roles for cytotoxic ...... versus noncytopathic viruses.
@en
P1476
Different roles for cytotoxic ...... versus noncytopathic viruses.
@en
P2093
P304
P356
10.1016/S0952-7915(96)80033-1
P577
1996-08-01T00:00:00Z