Differential activity of progesterone and glucocorticoid receptors on mouse mammary tumor virus templates differing in chromatin structure.
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The Mediator subunit MED1/TRAP220 is required for optimal glucocorticoid receptor-mediated transcription activationActivation of integrated provirus requires histone acetyltransferase. p300 and P/CAF are coactivators for HIV-1 TatMultiple Cbfa/AML sites in the rat osteocalcin promoter are required for basal and vitamin D-responsive transcription and contribute to chromatin organization.A ligand binding domain mutation in the mouse glucocorticoid receptor functionally links chromatin remodeling and transcription initiationChromosomal integration of retinoic acid response elements prevents cooperative transcriptional activation by retinoic acid receptor and retinoid X receptor.Dynamic access of the glucocorticoid receptor to response elements in chromatin.Progesterone and glucocorticoid receptors recruit distinct coactivator complexes and promote distinct patterns of local chromatin modification.Estrogen receptor-dependent proteasomal degradation of the glucocorticoid receptor is coupled to an increase in mdm2 protein expressionCofilin 1 is revealed as an inhibitor of glucocorticoid receptor by analysis of hormone-resistant cells.Glucocorticoid receptor domain requirements for chromatin remodeling and transcriptional activation of the mouse mammary tumor virus promoter in different nucleoprotein contexts.Interleukin-1-induced nuclear factor-kappaB-IkappaBalpha autoregulatory feedback loop in hepatocytes. A role for protein kinase calpha in post-transcriptional regulation of ikappabalpha resynthesis.Antiprogestins mediate differential effects on glucocorticoid receptor remodeling of chromatin structure.Progesterone receptor deficient in chromatin binding has an altered cellular state.The viral transactivator E1A regulates the mouse mammary tumor virus promoter in an isoform- and chromatin-specific manner.Nuclear receptor peroxisome proliferator-activated receptor alpha (PPARalpha) is expressed in resting murine lymphocytes. The PPARalpha in T and B lymphocytes is both transactivation and transrepression competent.Nuclear factor I-mediated repression of the mouse mammary tumor virus promoter is abrogated by the coactivators p300/CBP and SRC-1.Transcriptional regulation of mammalian genes in vivo. A tale of two templates.Transcription factor PU.1 controls transcription start site positioning and alternative TLR4 promoter usage.Transactivation specificity of glucocorticoid versus progesterone receptors. Role of functionally different interactions of transcription factors with amino- and carboxyl-terminal receptor domains.Identification and characterization of a tannic acid-responsive negative regulatory element in the mouse mammary tumor virus promoter.
P2860
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P2860
Differential activity of progesterone and glucocorticoid receptors on mouse mammary tumor virus templates differing in chromatin structure.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
Differential activity of proge ...... fering in chromatin structure.
@en
type
label
Differential activity of proge ...... fering in chromatin structure.
@en
prefLabel
Differential activity of proge ...... fering in chromatin structure.
@en
P2093
P2860
P356
P1476
Differential activity of proge ...... fering in chromatin structure.
@en
P2093
P2860
P304
14227-14235
P356
10.1074/JBC.272.22.14227
P407
P577
1997-05-01T00:00:00Z