Specific increase in p85alpha expression in response to dexamethasone is associated with inhibition of insulin-like growth factor-I stimulated phosphatidylinositol 3-kinase activity in cultured muscle cells.
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Increased insulin sensitivity in mice lacking p85beta subunit of phosphoinositide 3-kinaseReduced expression of the murine p85alpha subunit of phosphoinositide 3-kinase improves insulin signaling and ameliorates diabetesGSK-3beta activity is increased in skeletal muscle after burn injury in ratsDexamethasone modulates ErbB tyrosine kinase expression and signaling through multiple and redundant mechanisms in cultured rat hepatocytesAcute daily psychological stress causes increased atrophic gene expression and myostatin-dependent muscle atrophy.Diversification of cardiac insulin signaling involves the p85 alpha/beta subunits of phosphatidylinositol 3-kinase.Dual control of muscle cell survival by distinct growth factor-regulated signaling pathways.FOXO3a mediates signaling crosstalk that coordinates ubiquitin and atrogin-1/MAFbx expression during glucocorticoid-induced skeletal muscle atrophyGene expression during normal and FSHD myogenesis.4E-BP1 and S6K1: translational integration sites for nutritional and hormonal information in muscle.Muscle atrophy in chronic kidney disease results from abnormalities in insulin signaling.Molecular mechanisms of insulin resistance in type 2 diabetes mellitusThe sentrin-conjugating enzyme mUbc9 interacts with GLUT4 and GLUT1 glucose transporters and regulates transporter levels in skeletal muscle cellsAttenuated Pik3r1 expression prevents insulin resistance and adipose tissue macrophage accumulation in diet-induced obese mice.Early responses of insulin signaling to high-carbohydrate and high-fat overfeeding.11beta-hydroxysteroid dehydrogenase type 1 regulates glucocorticoid-induced insulin resistance in skeletal muscle.Prenatal stress and stress coping style interact to predict metabolic risk in male rats.Italian Society for the Study of Diabetes (SID)/Italian Endocrinological Society (SIE) guidelines on the treatment of hyperglycemia in Cushing's syndrome and acromegaly.Molecular Actions of Glucocorticoids in Cartilage and Bone During Health, Disease, and Steroid Therapy.Signal regulatory protein-α interacts with the insulin receptor contributing to muscle wasting in chronic kidney disease.Phosphoinositide 3-kinase/Akt signalling is responsible for the differential susceptibility of myoblasts and myotubes to menadione-induced oxidative stress.Early signal protein expression profiles in basophils: a population study.Insulin resistance and muscle metabolism in chronic kidney disease.Prolonged fasting and cortisol reduce myostatin mRNA levels in tilapia larvae; short-term fasting elevates.Inhibition of PI3-kinase signaling by glucocorticoids results in increased branched-chain amino acid degradation in renal epithelial cells.Phosphoinositide 3-kinase regulates myogenin expression at both the transcriptional and post-transcriptional level during myogenesis
P2860
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P2860
Specific increase in p85alpha expression in response to dexamethasone is associated with inhibition of insulin-like growth factor-I stimulated phosphatidylinositol 3-kinase activity in cultured muscle cells.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
Specific increase in p85alpha ...... vity in cultured muscle cells.
@en
type
label
Specific increase in p85alpha ...... vity in cultured muscle cells.
@en
prefLabel
Specific increase in p85alpha ...... vity in cultured muscle cells.
@en
P2093
P356
P1476
Specific increase in p85alpha ...... vity in cultured muscle cells.
@en
P2093
P304
P356
10.1074/JBC.272.11.7455
P407
P577
1997-03-01T00:00:00Z