about
Interaction of F1L with the BH3 domain of Bak is responsible for inhibiting vaccinia-induced apoptosisDirect reversal of glucocorticoid resistance by AKT inhibition in acute lymphoblastic leukemiaThe common feature of leukemia-associated IDH1 and IDH2 mutations is a neomorphic enzyme activity converting alpha-ketoglutarate to 2-hydroxyglutaratePTEN is a protein tyrosine phosphatase for IRS1ATP-citrate lyase links cellular metabolism to histone acetylationMetabolites produced by commensal bacteria promote peripheral regulatory T-cell generationToll-like receptor-induced changes in glycolytic metabolism regulate dendritic cell activationPraf2 is a novel Bcl-xL/Bcl-2 interacting protein with the ability to modulate survival of cancer cellsAsparagine plays a critical role in regulating cellular adaptation to glutamine depletionSerine catabolism regulates mitochondrial redox control during hypoxiaPrecision microbiome reconstitution restores bile acid mediated resistance to Clostridium difficile.Glutamine-based PET imaging facilitates enhanced metabolic evaluation of gliomas in vivoHypoxia promotes isocitrate dehydrogenase-dependent carboxylation of α-ketoglutarate to citrate to support cell growth and viabilityIntracellular crotonyl-CoA stimulates transcription through p300-catalyzed histone crotonylationIntestinal Blautia Is Associated with Reduced Death from Graft-versus-Host DiseaseHypoxia Induces Production of L-2-Hydroxyglutarate.Integration of 2-hydroxyglutarate-proton magnetic resonance spectroscopy into clinical practice for disease monitoring in isocitrate dehydrogenase-mutant glioma.The potential for isocitrate dehydrogenase mutations to produce 2-hydroxyglutarate depends on allele specificity and subcellular compartmentalization.Hypoxic and Ras-transformed cells support growth by scavenging unsaturated fatty acids from lysophospholipids.Arginase 1 is an innate lymphoid-cell-intrinsic metabolic checkpoint controlling type 2 inflammationCancer-associated IDH2 mutants drive an acute myeloid leukemia that is susceptible to Brd4 inhibition.Induction of sarcomas by mutant IDH2Carcinoma-astrocyte gap junctions promote brain metastasis by cGAMP transfer.Intracellular α-ketoglutarate maintains the pluripotency of embryonic stem cells.Interferon-γ regulates cellular metabolism and mRNA translation to potentiate macrophage activation.The X-linked mental retardation protein oligophrenin-1 is required for dendritic spine morphogenesis.Friends not foes: CTLA-4 blockade and mTOR inhibition cooperate during CD8+ T cell priming to promote memory formation and metabolic readiness.
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description
hulumtues
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Justin R. Cross
@ast
Justin R. Cross
@en
Justin R. Cross
@es
Justin R. Cross
@sl
type
label
Justin R. Cross
@ast
Justin R. Cross
@en
Justin R. Cross
@es
Justin R. Cross
@sl
prefLabel
Justin R. Cross
@ast
Justin R. Cross
@en
Justin R. Cross
@es
Justin R. Cross
@sl
P106
P1153
26639250300
P21
P31
P496
0000-0002-6356-057X