A "calcium set-point hypothesis" of neuronal dependence on neurotrophic factor.
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Cellular calcium deficiency plays a role in neuronal death caused by proteasome inhibitors.Cell-autonomous inhibition of alpha 7-containing nicotinic acetylcholine receptors prevents death of parasympathetic neurons during development.Up-regulation of the neuronal nicotinic receptor α7 by HIV glycoprotein 120: potential implications for HIV-associated neurocognitive disorderIntrinsically low open probability of α7 nicotinic acetylcholine receptors can be overcome by positive allosteric modulation and serum factors leading to the generation of excitotoxic currents at physiological temperaturesInhibition of electrical activity by retroviral infection with Kir2.1 transgenes disrupts electrical differentiation of motoneurons.Microarray analysis on human neuroblastoma cells exposed to aluminum, β(1-42)-amyloid or the β(1-42)-amyloid aluminum complex.Apoptosis and necrosis in cerebrovascular disease.Glutamate as a hippocampal neuron survival factor: an inherited defect in the trisomy 16 mouse.Time course of cell death due to acoustic overstimulation in the mouse medial geniculate body and primary auditory cortexAnoxia tolerant brains.Voltage-gated potassium channels at the crossroads of neuronal function, ischemic tolerance, and neurodegeneration.Downregulation of GluA2 AMPA receptor subunits reduces the dendritic arborization of developing spinal motoneurons.Cardiac glycosides provide neuroprotection against ischemic stroke: discovery by a brain slice-based compound screening platform.Functional dependence of Ca(2+)-activated K+ current on L- and N-type Ca2+ channels: differences between chicken sympathetic and parasympathetic neurons suggest different regulatory mechanisms.Implanted fibroblasts genetically engineered to produce brain-derived neurotrophic factor prevent 1-methyl-4-phenylpyridinium toxicity to dopaminergic neurons in the rat.Role of decreased sensory neuron membrane calcium currents in the genesis of neuropathic pain.Acute stimulation of transplanted neurons improves motoneuron survival, axon growth, and muscle reinnervation.Enhancing and regulating neurite outgrowth.Decline in age-dependent, MK801-induced injury coincides with developmental switch in parvalbumin expression: somatosensory and motor cortex.Brain tissue responses to ischemia.Effects of disrupting calcium homeostasis on neuronal maturation: early inhibition and later recoverySignaling pathways from cannabinoid receptor-1 activation to inhibition of N-methyl-D-aspartic acid mediated calcium influx and neurotoxicity in dorsal root ganglion neurons.Interleukin-10 release from astrocytes suppresses neuronal apoptosis via the TLR2/NFκB pathway in a neonatal rat model of hypoxic-ischemic brain damage.Abnormal calcium homeostasis in astrocytes from the trisomy 16 mouse.Neurotrophins and ciliary neurotrophic factor: their biology and pathology.Ex vivo imaging of active caspase 3 by a FRET-based molecular probe demonstrates the cellular dynamics and localization of the protease in cerebellar granule cells and its regulation by the apoptosis-inhibiting protein survivin.NMDAR blockade-induced neonatal brain injury: Reversal by the calcium channel agonist BayK 8644.Calmodulin mediates brain-derived neurotrophic factor cell survival signaling upstream of Akt kinase in embryonic neocortical neurons.NMDA antagonists exacerbate neuronal death caused by proteasome inhibition in cultured cortical and striatal neurons.Survival-promoting activity of nimodipine and nifedipine in rat motoneurons: implications of an intrinsic calcium toxicity in motoneurons.Akt activation is necessary for growth factor-induced trafficking of functional K(Ca) channels in developing parasympathetic neurons.Apoptotic mechanisms after repeated noise trauma in the mouse medial geniculate body and primary auditory cortex.Caspase-3 expression by cerebellar granule neurons is regulated by calcium and cyclic AMP.In vitro and in vivo neuroprotective activity of the cardiac glycoside oleandrin from Nerium oleander in brain slice-based stroke models.Role of cell-cell interactions in the developmental regulation of Ca2+-activated K+ currents in vertebrate neurons.
P2860
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P2860
A "calcium set-point hypothesis" of neuronal dependence on neurotrophic factor.
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
A "calcium set-point hypothesis" of neuronal dependence on neurotrophic factor.
@en
type
label
A "calcium set-point hypothesis" of neuronal dependence on neurotrophic factor.
@en
prefLabel
A "calcium set-point hypothesis" of neuronal dependence on neurotrophic factor.
@en
P2093
P1476
A "calcium set-point hypothesis" of neuronal dependence on neurotrophic factor.
@en
P2093
Franklin J
Johnson EM Jr
P304
P356
10.1016/0014-4886(92)90242-I
P407
P577
1992-01-01T00:00:00Z