Reducing sugars trigger oxidative modification and apoptosis in pancreatic beta-cells by provoking oxidative stress through the glycation reaction
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Spotlight on ethnomedicine: usability of Sutherlandia frutescens in the treatment of diabetesStudies on the ADP-ribose pyrophosphatase subfamily of the nudix hydrolases and tentative identification of trgB, a gene associated with tellurite resistance.Thioredoxins, glutaredoxins, and peroxiredoxins--molecular mechanisms and health significance: from cofactors to antioxidants to redox signalingAbsence of a reductase, NCB5OR, causes insulin-deficient diabetesLiraglutide improves pancreatic Beta cell mass and function in alloxan-induced diabetic miceProtective effects of fluvastatin against reactive oxygen species induced DNA damage and mutagenesis.Down regulation of superoxide dismutases and glutathione peroxidase by reactive oxygen and nitrogen species.Nutritionally induced insulin resistance and receptor defect leading to beta-cell failure in animal models.Oxidative stress, nitric oxide, and diabetes.A role for glutathione peroxidase in protecting pancreatic beta cells against oxidative stress in a model of glucose toxicity.Padina arborescens extract protects high glucose-induced apoptosis in pancreatic β cells by reducing oxidative stress.Small-molecule inhibition of inflammatory β-cell death.Oral administration of ginseng ameliorates cyclosporine-induced pancreatic injury in an experimental mouse model.Nucleotide pyrophosphatase/phosphodiesterase 1 exerts a negative effect on starch accumulation and growth in rice seedlings under high temperature and CO2 concentration conditions.Advanced glycation end products are direct modulators of β-cell function.Prevention of glucose toxicity in HIT-T15 cells and Zucker diabetic fatty rats by antioxidants.Relationship of Soluble RAGE with Insulin Resistance and Beta Cell Function during Development of Type 2 Diabetes Mellitus.The production of cross-reactive autoantibodies that bind to bovine serum albumin in mice administered reducing sugars by subcutaneous injection.Advanced-glycation end products in insulin-resistant states.Nutritionally induced diabetes in desert rodents as models of type 2 diabetes: Acomys cahirinus (spiny mice) and Psammomys obesus (desert gerbil).Dietary Advanced Glycation End Products Consumption as a Direct Modulator of Insulin Sensitivity in Overweight Humans: A Study Protocol for a Double-Blind, Randomized, Two Period Cross-Over Trial.Glucolipotoxicity: fuel excess and beta-cell dysfunction.Genetic disruption of SOD1 gene causes glucose intolerance and impairs β-cell function.Oxidative stress in diabetes: implications for vascular and other complications.Apoptosis in insulin-secreting cells. Evidence for the role of intracellular Ca2+ stores and arachidonic acid metabolism.Effects of soybean isoflavone extract on the plasma lipid profiles and antioxidant enzyme activity in streptozotocin-induced diabetic rats.Regulatory landscape of AGE-RAGE-oxidative stress axis and its modulation by PPARγ activation in high fructose diet-induced metabolic syndrome.Problems associated with glucose toxicity: role of hyperglycemia-induced oxidative stress.Regulation of insulin synthesis and secretion and pancreatic Beta-cell dysfunction in diabetes.Diabetes, oxidative stress and physical exercise.Role of reactive oxygen species in the progression of type 2 diabetes and atherosclerosis.Oxidative stress and beta-cell dysfunction.Oxidative and non-oxidative DNA damage and cardiovascular disease.Involvement of oxidative stress in suppression of insulin biosynthesis under diabetic conditions.Glucagon: the renewal of an old hormone in the pathophysiology of diabetes.Down-regulation of pancreatic transcription factors and incretin receptors in type 2 diabetes.Therapeutic effects of sericin on diabetic keratopathy in Otsuka Long-Evans Tokushima Fatty rats.Role of pancreatic transcription factors in maintenance of mature β-cell function.Extracts of Chrysanthemum zawadskii attenuate oxidative damage to vascular endothelial cells caused by a highly reducing sugar.Phloroglucinol Protects INS-1 Pancreatic β-cells Against Glucotoxicity-Induced Apoptosis.
P2860
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P2860
Reducing sugars trigger oxidative modification and apoptosis in pancreatic beta-cells by provoking oxidative stress through the glycation reaction
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Reducing sugars trigger oxidat ...... through the glycation reaction
@en
type
label
Reducing sugars trigger oxidat ...... through the glycation reaction
@en
prefLabel
Reducing sugars trigger oxidat ...... through the glycation reaction
@en
P2093
P2860
P356
P1433
P1476
Reducing sugars trigger oxidat ...... through the glycation reaction
@en
P2093
Kawasaki Y
Miyazawa N
Nakamura M
Taniguchi N
P2860
P304
P356
10.1042/BJ3200855
P407
P478
320 ( Pt 3)
P577
1996-12-01T00:00:00Z