Microglial TNF-α Suppresses Cocaine-Induced Plasticity and Behavioral Sensitization.
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Neuron-Microglia Interactions in Mental Health Disorders: "For Better, and For Worse"Activin A is increased in the nucleus accumbens following a cocaine bingeMicroglial depletion alters the brain neuroimmune response to acute binge ethanol withdrawalA metaplasticity view of the interaction between homeostatic and Hebbian plasticity.The interplay between neurons and glia in synapse development and plasticity.l-DOPA-induced dyskinesia and neuroinflammation: do microglia and astrocytes play a role?Opioid Self-Administration is Attenuated by Early-Life Experience and Gene Therapy for Anti-Inflammatory IL-10 in the Nucleus Accumbens of Male Rats.Novel Influences of IL-10 on CNS Inflammation Revealed by Integrated Analyses of Cytokine Networks and Microglial Morphology.Local Cues Establish and Maintain Region-Specific Phenotypes of Basal Ganglia Microglia.Roles of Glial Cells in Sculpting Inhibitory Synapses and Neural Circuits.Deprivation-Induced Homeostatic Spine Scaling In Vivo Is Localized to Dendritic Branches that Have Undergone Recent Spine Loss.Alcohol and Stress Activation of Microglia and Neurons: Brain Regional Effects.Alzheimer's Disease: The Role of Microglia in Brain Homeostasis and Proteopathy.Microglia Gone Rogue: Impacts on Psychiatric Disorders across the Lifespan.Inflammation alters AMPA-stimulated calcium responses in dorsal striatal D2 but not D1 spiny projection neurons.Toll-like receptor 4 deficiency alters nucleus accumbens synaptic physiology and drug reward behavior.Granulocyte-colony stimulating factor controls neural and behavioral plasticity in response to cocaine.A microglia-cytokine axis to modulate synaptic connectivity and function.Cafeteria diet induces neuroplastic modifications in the nucleus accumbens mediated by microglia activation.Cocaine-Mediated Downregulation of miR-124 Activates Microglia by Targeting KLF4 and TLR4 Signaling.Chemokines and cocaine: CXCR4 receptor antagonist AMD3100 attenuates cocaine place preference and locomotor stimulation in rats.Tumour necrosis factor-mediated homeostatic synaptic plasticity in behavioural models: testing a role in maternal immune activation.Chemokines and 'bath salts': CXCR4 receptor antagonist reduces rewarding and locomotor-stimulant effects of the designer cathinone MDPV in rats.Toll-like receptor 3 modulates the behavioral effects of cocaine in mice.Lung-injury depresses glutamatergic synaptic transmission in the nucleus tractus solitarii via discrete age-dependent mechanisms in neonatal rats.Beyond simple tests of value: measuring addiction as a heterogeneous disease of computation-specific valuation processesTNF-α increases the intrinsic excitability of cerebellar Purkinje cells through elevating glutamate release in Bergmann Glia
P2860
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P2860
Microglial TNF-α Suppresses Cocaine-Induced Plasticity and Behavioral Sensitization.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Microglial TNF-α Suppresses Cocaine-Induced Plasticity and Behavioral Sensitization.
@en
type
label
Microglial TNF-α Suppresses Cocaine-Induced Plasticity and Behavioral Sensitization.
@en
prefLabel
Microglial TNF-α Suppresses Cocaine-Induced Plasticity and Behavioral Sensitization.
@en
P2093
P2860
P1433
P1476
Microglial TNF-α Suppresses Cocaine-Induced Plasticity and Behavioral Sensitization.
@en
P2093
Andrew D Greenhalgh
David Stellwagen
Gil M Lewitus
Horia Pribiag
Keanan Augereau
Sarah C Konefal
P2860
P304
P356
10.1016/J.NEURON.2016.03.030
P407
P577
2016-04-20T00:00:00Z