about
Therapeutic approach to regulate innate immune response by Toll-like receptor 4 antagonist E5564 in rats with D-galactosamine-induced acute severe liver injuryFluorescent In situ hybridization allows rapid identification of microorganisms in blood cultures.LL-37 protects rats against lethal sepsis caused by gram-negative bacteriaChildhood septicemia in Nepal: documenting the bacterial etiology and its susceptibility to antibiotics.Receptors, mediators, and mechanisms involved in bacterial sepsis and septic shock.Down-regulation of Stathmin Is Required for the Phenotypic Changes and Classical Activation of Macrophages.The birth pangs of monoclonal antibody therapeutics: the failure and legacy of CentoxinDirect identification of bacteria from positive blood cultures by amplification and sequencing of the 16S rRNA gene: evaluation of BACTEC 9240 instrument true-positive and false-positive resultsCharacterization of a monoclonal antibody that binds to an epitope on soluble bacterial peptidoglycan fragments.1,25-Dihydroxyvitamin D inhibits lipopolysaccharide-induced immune activation in human endothelial cells.Bacteremia in children at the University Hospital in Riyadh, Saudi Arabia.Pathobiology of lipopolysaccharide.The phosphatidylinositol 3-kinase pathway selectively controls sIL-1RA not interleukin-1beta production in the septic leukocytes.Reduced oxidative tissue damage during endotoxemia in IRAK-1 deficient miceSepsis and polyspecific intravenous immunoglobulins.Bacterial lipopolysaccharide activates nuclear factor-kappaB through interleukin-1 signaling mediators in cultured human dermal endothelial cells and mononuclear phagocytes.Bacterial lipopolysaccharide activates NF-kappaB through toll-like receptor 4 (TLR-4) in cultured human dermal endothelial cells. Differential expression of TLR-4 and TLR-2 in endothelial cells.Gelam honey has a protective effect against lipopolysaccharide (LPS)-induced organ failure.Oxidative DNA damage and total antioxidant status in rats during experimental gram-negative sepsis.Ketamine suppresses intestinal NF-kappa B activation and proinflammatory cytokine in endotoxic rats.Cathelicidin peptide sheep myeloid antimicrobial peptide-29 prevents endotoxin-induced mortality in rat models of septic shock.Effects of Porphyromonas gingivalis antigens and proinflammatory cytokines on human coronary artery endothelial cells.Differential cytokine response in host defence mechanisms triggered by gram-negative and gram-positive bacteria, and the roles of gabexate mesilate, a synthetic protease inhibitor.The effect of methylprednisolone on treatment in rats with induced sepsis.Cecropin B enhances betalactams activities in experimental rat models of gram-negative septic shock.Ketamine suppresses endotoxin-induced NF-kappaB activation and cytokines production in the intestine.The prophylactic effects of natural water-soluble antioxidant from spinach and apocynin in a rabbit model of lipopolysaccharide-induced endotoxemia.Complexity of inflammatory responses in endothelial cells and vascular smooth muscle cells determined by microarray analysis.
P2860
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P2860
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Current understanding of sepsis.
@en
type
label
Current understanding of sepsis.
@en
prefLabel
Current understanding of sepsis.
@en
P2093
P356
P1476
Current understanding of sepsis.
@en
P2093
P304
P356
10.1093/CLINIDS/22.3.407
P407
P577
1996-03-01T00:00:00Z