The cytotoxicity of tumor necrosis factor depends on induction of the mitochondrial permeability transition.
about
Adenine nucleotide (ADP/ATP) translocase 3 participates in the tumor necrosis factor induced apoptosis of MCF-7 cellsInhibition of receptor internalization by monodansylcadaverine selectively blocks p55 tumor necrosis factor receptor death domain signalingCytochrome c release upon Fas receptor activation depends on translocation of full-length bid and the induction of the mitochondrial permeability transitionColchicine protects mice from the lethal effect of an agonistic anti-Fas antibodyBcl-2 resistant mitochondrial toxicity mediated by the isoquinoline carboxamide PK11195 involves de novo generation of reactive oxygen speciesLow circulating levels of dehydroepiandrosterone in histologically advanced nonalcoholic fatty liver diseaseControlling the mitochondrial gatekeeper for effective chemotherapy.Tumor necrosis factor-related apoptosis-inducing ligand alters mitochondrial membrane lipids.Hexokinase II binding to mitochondria is necessary for Kupffer cell activation and is potentiated by ethanol exposureEthanol sensitizes mitochondria to the permeability transition by inhibiting deacetylation of cyclophilin-D mediated by sirtuin-3Cathepsin B contributes to TNF-alpha-mediated hepatocyte apoptosis by promoting mitochondrial release of cytochrome cMechanisms of organ dysfunction in critical illness: report from a Round Table Conference held in Brussels.Alcohol and mitochondria: a dysfunctional relationshipNew advances on structural and biological functions of ceramide in apoptotic/necrotic cell death and cancer.Oxidative stress in glaucomatous neurodegeneration: mechanisms and consequences.Blocking mPTP on Neural Stem Cells and Activating the Nicotinic Acetylcholine Receptor α7 Subunit on Microglia Attenuate Aβ-Induced Neurotoxicity on Neural Stem Cells.Prevention of experimental allergic encephalomyelitis by targeting nitric oxide and peroxynitrite: implications for the treatment of multiple sclerosisThe myxoma poxvirus protein, M11L, prevents apoptosis by direct interaction with the mitochondrial permeability transition pore.Lipopolysaccharide induces disseminated endothelial apoptosis requiring ceramide generation.The permeability transition pore complex: a target for apoptosis regulation by caspases and bcl-2-related proteins.Sirtuin-4 modulates sensitivity to induction of the mitochondrial permeability transition pore.Sepsis: links between pathogen sensing and organ damageCeramide and mitochondria in ischemia/reperfusion.Oxyphenisatin acetate (NSC 59687) triggers a cell starvation response leading to autophagy, mitochondrial dysfunction, and autocrine TNFα-mediated apoptosis.Mitochondria and redox signaling in steatohepatitisCeramide and mitochondria in ischemic brain injury.Therapeutic mechanisms of single Chinese medicine herb or their extracts for extrahepatic obstructive jaundice.Mitochondrial channels: ion fluxes and more.Sensitization of ionizing radiation-induced apoptosis by ursolic acid.Suppression of mitochondrial NADP(+)-dependent isocitrate dehydrogenase activity enhances curcumin-induced apoptosis in HCT116 cells.Metaxin deficiency alters mitochondrial membrane permeability and leads to resistance to TNF-induced cell killingKnockdown of sensitive to apoptosis gene by small interfering RNA enhances the sensitivity of PC3 cells toward actinomycin D and etoposide.Mitochondria-targeted antioxidants do not prevent tumour necrosis factor-induced necrosis of L929 cells.Diazepam enhances etoposide-induced cytotoxicity in U-87 MG human glioma cell line.Cytochrome c-dependent activation of caspase-3 by tumor necrosis factor requires induction of the mitochondrial permeability transition.Coenzyme Q10 can in some circumstances block apoptosis, and this effect is mediated through mitochondria.Aryl hydrocarbon receptor regulation of ceramide-induced apoptosis in murine hepatoma 1c1c7 cells. A function independent of aryl hydrocarbon receptor nuclear translocator.Signal transduction of stress via ceramide.Oligomeric BAX induces mitochondrial permeability transition and complete cytochrome c release without oxidative stress.Mitochondrial disorders in NSAIDs-induced small bowel injury.
P2860
Q24672726-1BCCD178-2CE5-4945-82E9-D0BC8401DB91Q28140492-6A1E12A5-209F-403E-B716-06A43FE0E358Q28215534-14A72BF5-D0DB-4271-8F7F-71C0EBA40259Q28343905-2BB4E096-108A-4687-B485-B9C795EE5878Q28365278-191E827B-63D9-4DC2-BB5A-441ADF5954D3Q34003358-329960DF-7BBB-43F9-AF66-CA919093F32FQ34089134-E1173F05-1BB6-45E7-8D9B-925EA7EB872AQ34139591-BAB7D6D9-7A3A-4785-AA3C-32E079D341F8Q34249104-20DD507E-2D68-4552-BE02-774F34C5AAFDQ34329593-32099B3B-7645-42A7-8D4B-5332A4FBB9B0Q34457741-67BA513E-22DC-4EE9-A60C-DF1B7C664FE5Q34571280-D8BD55D7-B1D4-48E6-8989-283B784CEDCFQ34676867-19B73B41-73AC-4A49-B651-8B1092144709Q34975049-12E8B059-F5FC-4E33-A40A-454ACAB193F5Q35677091-B139B4BE-6FEB-4BB2-B3C0-DDFAC4E625D0Q35923426-4F95229D-549E-461D-B8DB-F48C24FC08D3Q36049347-ECBF77A4-5C6A-4D1A-A840-9F096A25F02DQ36371468-7A4F79F1-A08E-4747-B952-B5754FB751ABQ36380942-156C39EB-4597-4B54-870B-6B423CD036D6Q36400661-206973F8-C772-4632-9E5F-C1D96868D85BQ36401146-F4A51FF5-E9B2-48C4-915E-FD7869CC6E1FQ37239817-DA9C1012-4B3F-45B9-ABF2-56119CAAC1E1Q37344754-B73C455D-0086-4A7C-84DD-8887F02BA67AQ37482581-72845BE1-41EF-4F86-AE20-EF9D36B2C44EQ37815476-E77EF1AE-97C4-48D3-A61D-CC2D0D555CDDQ37970353-99E96D34-FEAD-45C5-8712-4ECBB1F0F0C3Q38182949-38CA2220-5B27-4E17-8F4B-DCEB0A08C633Q38201407-0505C0BB-977E-4A61-A4AB-166365CB58BDQ39413290-50528988-882F-4BAE-A3AD-550A5FA89F4CQ39626958-1F6A3932-81F5-49D9-B443-1839BE197341Q39629983-B37D4C90-399B-4FC4-9EE0-5B7E85FA1B66Q39696218-69EC0770-4FDC-48ED-83B4-DA588887CDB8Q40087541-D1E8C503-44B1-40AE-B592-D104EB08417FQ40790543-206E9CFB-BA4B-45B8-BC07-F5FCE3EF0C6EQ40873865-408E6253-ED07-45DA-9CE9-2BDF2A857200Q40900880-CD2D4C55-E204-4518-A852-642DE56B79D6Q40980110-CAEED37D-00DB-4E99-B4F1-1C7F07AFDC9AQ41841345-E7DB3239-05A8-463E-A180-DBC7F32212ABQ41980121-4CFD25F8-51CA-4FBE-97C0-EE73F83E5829Q42015382-D97BD3E4-C026-4DD3-AF8A-9F5164CFEC5D
P2860
The cytotoxicity of tumor necrosis factor depends on induction of the mitochondrial permeability transition.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
The cytotoxicity of tumor necr ...... drial permeability transition.
@en
type
label
The cytotoxicity of tumor necr ...... drial permeability transition.
@en
prefLabel
The cytotoxicity of tumor necr ...... drial permeability transition.
@en
P2093
P356
P1476
The cytotoxicity of tumor necr ...... drial permeability transition.
@en
P2093
Glascott PA Jr
Pastorino JG
Rothman RJ
Yamamoto K
P304
29792-29798
P356
10.1074/JBC.271.47.29792
P407
P577
1996-11-01T00:00:00Z