Dysferlin deficiency confers increased susceptibility to coxsackievirus-induced cardiomyopathy.

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Preventing pediatric cardiomyopathy: a 2015 outlook.Phosphorylation and degradation of αB-crystallin during enterovirus infection facilitates viral replication and induces viral pathogenesis.

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Dysferlin deficiency confers increased susceptibility to coxsackievirus-induced cardiomyopathy.

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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name

Dysferlin deficiency confers i ...... evirus-induced cardiomyopathy.

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type

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Dysferlin deficiency confers i ...... evirus-induced cardiomyopathy.

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Dysferlin deficiency confers i ...... evirus-induced cardiomyopathy.

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Dysferlin deficiency confers i ...... evirus-induced cardiomyopathy.

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Chen Wang

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Gabriel Fung

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Haoyu Deng

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Honglin Luo

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Jerry Wong

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Jingchun Zhang

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Junyan Shi

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Pascal Bernatchez

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P2860

Utrophin-dystrophin-deficient mice as a model for Duchenne muscular dystrophy

Defective membrane repair in dysferlin-deficient muscular dystrophy

Cloning of the mouse dysferlin gene and genomic characterization of the SJL-Dysf mutation

Dysferlin, a novel skeletal muscle gene, is mutated in Miyoshi myopathy and limb girdle muscular dystrophy

Early detection of cardiac involvement in Miyoshi myopathy: 2D strain echocardiography and late gadolinium enhancement cardiovascular magnetic resonance

Dysferlin and the plasma membrane repair in muscular dystrophy.

Enteroviral protease 2A cleaves dystrophin: evidence of cytoskeletal disruption in an acquired cardiomyopathy.

Molecular pathology of inflammatory cardiomyopathy.

Cleavage of serum response factor mediated by enteroviral protease 2A contributes to impaired cardiac function

Dysferlin-mediated membrane repair protects the heart from stress-induced left ventricular injury.

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1423-1430

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scholarly article

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10.1111/CMI.12473

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2015-06-15T00:00:00Z

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26073173

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Coxsackievirus

Dysferlin deficiency confers increased susceptibility to coxsackievirus-induced cardiomyopathy.

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P2860

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Dysferlin deficiency confers increased susceptibility to coxsackievirus-induced cardiomyopathy.

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