Enterococcus faecalis induces aneuploidy and tetraploidy in colonic epithelial cells through a bystander effect.
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Oxidative stress in cancer associated fibroblasts drives tumor-stroma co-evolution: A new paradigm for understanding tumor metabolism, the field effect and genomic instability in cancer cellsHelicobacter pylori and colorectal neoplasia: Is there a causal link?Relationship between intestinal microbiota and colorectal cancerRole of the Microbiota in Colorectal Cancer: Updates on Microbial Associations and Therapeutic ImplicationsCancer chemoprevention: Much has been done, but there is still much to do. State of the art and possible new approachesColon Macrophages Polarized by Commensal Bacteria Cause Colitis and Cancer through the Bystander Effect.Increased cancer risk of augmentation cystoplasty: possible role for hyperosmolal microenvironment on DNA damage recognitionGut microbial diversity in health and disease: experience of healthy Indian subjects, and colon carcinoma and inflammatory bowel disease patients.Bacterial oncogenesis in the colonTowards the human colorectal cancer microbiome.Detection of human intestinal catalase-negative, Gram-positive cocci by rRNA-targeted reverse transcription-PCR.p53 dependent centrosome clustering prevents multipolar mitosis in tetraploid cellsEnterococcus faecalis infection causes inflammation, intracellular oxphos-independent ROS production, and DNA damage in human gastric cancer cells.A selenium-dependent xanthine dehydrogenase triggers biofilm proliferation in Enterococcus faecalis through oxidant production.Microbiota disbiosis is associated with colorectal cancer.Microbes, microbiota, and colon cancer.Commensal bacteria drive endogenous transformation and tumour stem cell marker expression through a bystander effect.Quantitative profiling of colorectal cancer-associated bacteria reveals associations between fusobacterium spp., enterotoxigenic Bacteroides fragilis (ETBF) and clinicopathological features of colorectal cancer.Microbiota and immune responses in colon cancer: more to learn.Illicit survival of cancer cells during polyploidization and depolyploidization.Hydrogen peroxide fuels aging, inflammation, cancer metabolism and metastasis: the seed and soil also needs "fertilizer".Understanding Luminal Microorganisms and Their Potential Effectiveness in Treating Intestinal InflammationColorectal cancer: role of commensal bacteria and bystander effects.The struggle within: microbial influences on colorectal cancer.Tetraploid cells from cytokinesis failure induce aneuploidy and spontaneous transformation of mouse ovarian surface epithelial cells.Bacterially-Associated Transcriptional Remodelling in a Distinct Genomic Subtype of Colorectal Cancer Provides a Plausible Molecular Basis for Disease Development.Enterococcus faecalis enhances cell proliferation through hydrogen peroxide-mediated epidermal growth factor receptor activationTNF-α mediates macrophage-induced bystander effects through Netrin-1.Helicobacter pylori infection and colorectal carcinoma: is there a causal association?Colorectal carcinoma: Importance of colonic environment for anti-cancer response and systemic immunity.On the origin of reactive oxygen species and antioxidative mechanisms in Enterococcus faecalis.Beneficial effects of lactic acid bacteria on human beings.The role of gut microbiota in the pathogenesis of colorectal cancer.Cancer-associated fibroblasts connect metastasis-promoting communication in colorectal cancer.Phenotype transformation of immortalized NCM460 colon epithelial cell line by TGF-β1 is associated with chromosome instability.Gut Microbiota, Inflammation, and Colorectal Cancer.Enterococcus faecalis Infection and Reactive Oxygen Species Down-Regulates the miR-17-92 Cluster in Gastric Adenocarcinoma Cell Culture.Infectious Endocarditis from Enterococcus faecalis Associated with Tubular Adenoma of the Sigmoid Colon.The coffee-machine bacteriome: biodiversity and colonisation of the wasted coffee tray leach.Cyclooxygenase-2 generates the endogenous mutagen trans-4-hydroxy-2-nonenal in Enterococcus faecalis-infected macrophages.
P2860
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P2860
Enterococcus faecalis induces aneuploidy and tetraploidy in colonic epithelial cells through a bystander effect.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Enterococcus faecalis induces ...... ls through a bystander effect.
@en
type
label
Enterococcus faecalis induces ...... ls through a bystander effect.
@en
prefLabel
Enterococcus faecalis induces ...... ls through a bystander effect.
@en
P2093
P2860
P1433
P1476
Enterococcus faecalis induces ...... ls through a bystander effect.
@en
P2093
Courtney W Houchen
Mark M Huycke
Randal J May
Stanley Lightfoot
Toby D Allen
Xingmin Wang
P2860
P304
P356
10.1158/0008-5472.CAN-08-1551
P407
P577
2008-12-01T00:00:00Z