Production of tumor necrosis factor-alpha as a result of glia-T-cell interaction correlates with the pathogenic activity of myelin basic protein-reactive T cells in experimental autoimmune encephalomyelitis.
about
Pharmacological characterization of novel A3 adenosine receptor-selective antagonistsMajor histocompatibility complex molecules on parenchymal cells of the target organ protect against autoimmune disease.Retinal astrocytes pretreated with NOD2 and TLR2 ligands activate uveitogenic T cells.Glaucomatous tissue stress and the regulation of immune response through glial Toll-like receptor signalingMechanisms of immune system activation in glaucoma: oxidative stress-stimulated antigen presentation by the retina and optic nerve head glia
P2860
Production of tumor necrosis factor-alpha as a result of glia-T-cell interaction correlates with the pathogenic activity of myelin basic protein-reactive T cells in experimental autoimmune encephalomyelitis.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Production of tumor necrosis f ...... autoimmune encephalomyelitis.
@en
type
label
Production of tumor necrosis f ...... autoimmune encephalomyelitis.
@en
prefLabel
Production of tumor necrosis f ...... autoimmune encephalomyelitis.
@en
P2093
P1476
Production of tumor necrosis f ...... autoimmune encephalomyelitis.
@en
P2093
P304
P356
10.1002/(SICI)1097-4547(19960815)45:4<400::AID-JNR9>3.0.CO;2-D
P577
1996-08-01T00:00:00Z