In vivo studies of cartilage regeneration after damage induced by catabolin/interleukin-1.
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The triterpenoid CDDO inhibits expression of matrix metalloproteinase-1, matrix metalloproteinase-13 and Bcl-3 in primary human chondrocytesDifferential recovery of glycosaminoglycan after IL-1-induced degradation of bovine articular cartilage depends on degree of degradationEsculetin inhibits cartilage resorption induced by interleukin 1alpha in combination with oncostatin MThe role of cytokines as inflammatory mediators in osteoarthritis: lessons from animal models.Variations in the TNFα gene and their interactions with the IL4R and IL10 genes in relation to hand osteoarthritis.Changes in serum and synovial fluid biomarkers after acute injury (NCT00332254).Aspartic acid racemization reveals a high turnover state in knee compared with hip osteoarthritic cartilage.Composition-function relationships during IL-1-induced cartilage degradation and recovery.Analysing the role of endogenous matrix molecules in the development of osteoarthritis.Is Synovial Macrophage Activation the Inflammatory Link Between Obesity and Osteoarthritis?Interplay between viruses and bacterial microbiota in cancer development.Synergistic induction of matrix metalloproteinase 1 by interleukin-1alpha and oncostatin M in human chondrocytes involves signal transducer and activator of transcription and activator protein 1 transcription factors via a novel mechanism.Synergistic effects of glycoprotein 130 binding cytokines in combination with interleukin-1 on cartilage collagen breakdown.Morphological changes of bovine nasal chondrocytes induced by interleukin-1αBlocking cartilage destruction with metalloproteinase inhibitors: a valid therapeutic target?Effects of interleukin-1 beta on insulin-like growth factor-I autocrine/paracrine axis in cultured rat articular chondrocytesEffect of tenidap on cartilage integrity in vitro.Interleukin 17 induces cartilage collagen breakdown: novel synergistic effects in combination with proinflammatory cytokines.Interleukin 13 blocks the release of collagen from bovine nasal cartilage treated with proinflammatory cytokines.Human nasal cartilage responds to oncostatin M in combination with interleukin 1 or tumour necrosis factor alpha by the release of collagen fragments via collagenases.Activation of procollagenases is a key control point in cartilage collagen degradation: interaction of serine and metalloproteinase pathways.A novel paradigm for dendritic cells as effectors of cartilage destruction.Comparison of lesions induced by intra-articular injections of quinolones and compounds damaging cartilage components in rat femoral condyles.Effects of salmon DNA fraction in vitro and in a monosodium iodoacetate-induced osteoarthritis rat model.
P2860
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P2860
In vivo studies of cartilage regeneration after damage induced by catabolin/interleukin-1.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
In vivo studies of cartilage r ...... ed by catabolin/interleukin-1.
@en
type
label
In vivo studies of cartilage r ...... ed by catabolin/interleukin-1.
@en
prefLabel
In vivo studies of cartilage r ...... ed by catabolin/interleukin-1.
@en
P2093
P2860
P356
P1476
In vivo studies of cartilage r ...... ed by catabolin/interleukin-1.
@en
P2093
Page Thomas DP
Stephens T
P2860
P356
10.1136/ARD.50.2.75
P407
P577
1991-02-01T00:00:00Z