Selective inhibition of intra-alveolar p55 TNF receptor attenuates ventilator-induced lung injury. - Wikidata - awgldk - TriplyDB

Selective inhibition of intra-alveolar p55 TNF receptor attenuates ventilator-induced lung injury.

Selective inhibition of intra-alveolar p55 TNF receptor attenuates ventilator-induced lung injury.

http://www.wikidata.org/entity/Q41252465

about

Role of the type I tumor necrosis factor receptor in inflammation-associated olfactory dysfunction.Membrane translocation of IL-33 receptor in ventilator induced lung injury Monocyte Tumor Necrosis Factor-α-Converting Enzyme Catalytic Activity and Substrate Shedding in Sepsis and Noninfectious Systemic Inflammation.Lung epithelial cell-derived extracellular vesicles activate macrophage-mediated inflammatory responses via ROCK1 pathway.TNF-induced death signaling triggers alveolar epithelial dysfunction in acute lung injury.The double macchiato years; awards for the best basic science and epidemiology papers in 2012.Biological therapies in the acute respiratory distress syndrome.The Lectin-like Domain of TNF Increases ENaC Open Probability through a Novel Site at the Interface between the Second Transmembrane and C-terminal Domains of the α-Subunit Specificity of human anti-variable heavy (VH ) chain autoantibodies and impact on the design and clinical testing of a VH domain antibody antagonist of tumour necrosis factor-α receptor 1.Inhibition of TNF Receptor p55 By a Domain Antibody Attenuates the Initial Phase of Acid-Induced Lung Injury in Mice.Aspirin reduces lipopolysaccharide-induced pulmonary inflammation in human models of ARDS.Endothelial cell signaling and ventilator-induced lung injury: molecular mechanisms, genomic analyses, and therapeutic targets.Future therapies for ARDS.Anti-TNFα therapy in inflammatory lung diseases.High-Fat Feeding Protects Mice From Ventilator-Induced Lung Injury, Via Neutrophil-Independent Mechanisms.Kinetic profiling of in vivo lung cellular inflammatory responses to mechanical ventilation.Ventilation with "clinically relevant" high tidal volumes does not promote stretch-induced injury in the lungs of healthy mice.Atelectasis causes alveolar hypoxia-induced inflammation during uneven mechanical ventilation in rats Inflammatory mechanisms of ventilator-induced lung injury: a time to stop and think?The effect of etanercept on lung leukocyte margination and fibrin deposition after cardiac surgery.Stability-Diversity Tradeoffs Impose Fundamental Constraints on Selection of Synthetic Human VH/VL Single-Domain Antibodies from In Vitro Display Libraries.A New Venue of TNF Targeting.

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Selective inhibition of intra-alveolar p55 TNF receptor attenuates ventilator-induced lung injury.

http://www.wikidata.org/entity/Q41252465

description

2011 nî lūn-bûn

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年學術文章

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name

Selective inhibition of intra- ...... entilator-induced lung injury.

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Selective inhibition of intra- ...... entilator-induced lung injury.

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THORAXJNL-2011-200590

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Selective inhibition of intra- ...... entilator-induced lung injury.

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Andrew Bayliffe

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Masao Takata

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Michael R Wilson

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Peter J Morley

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Ruud de Wildt

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Szabolcs Bertok

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P2860

Etk/Bmx as a tumor necrosis factor receptor type 2-specific kinase: role in endothelial cell migration and angiogenesis

Ventilation with lower tidal volumes as compared with traditional tidal volumes for acute lung injury and the acute respiratory distress syndrome. The Acute Respiratory Distress Syndrome Network

Domain antibodies: proteins for therapy

Efficacy and safety of the monoclonal anti-tumor necrosis factor antibody F(ab')2 fragment afelimomab in patients with severe sepsis and elevated interleukin-6 levels

Efficacy and safety of inhaled carbon monoxide during pulmonary inflammation in mice

Binding activities of a repertoire of single immunoglobulin variable domains secreted from Escherichia coli.

TNF receptors differentially signal and are differentially expressed and regulated in the human heart.

Linking lung function and inflammatory responses in ventilator-induced lung injury.

Causes and timing of death in patients with ARDS.

Elevated plasma levels of soluble TNF receptors are associated with morbidity and mortality in patients with acute lung injury.

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