Insulin-stimulated GLUT4 translocation is mediated by a divergent intracellular signaling pathway.
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Interaction of insulin receptor substrate-1 with the sigma3A subunit of the adaptor protein complex-3 in cultured adipocytesSKIP negatively regulates insulin-induced GLUT4 translocation and membrane ruffle formation.PtdIns-specific MPR pathway association of a novel WD40 repeat protein, WIPI49Association of the insulin receptor with phospholipase C-gamma (PLCgamma) in 3T3-L1 adipocytes suggests a role for PLCgamma in metabolic signaling by insulinKinase activation through dimerization by human SH2-BOverexpression of SH2-containing inositol phosphatase 2 results in negative regulation of insulin-induced metabolic actions in 3T3-L1 adipocytes via its 5'-phosphatase catalytic activityChronic endothelin-1 treatment leads to heterologous desensitization of insulin signaling in 3T3-L1 adipocytesbeta -Arrestin-mediated recruitment of the Src family kinase Yes mediates endothelin-1-stimulated glucose transportInsulin increases the association of Akt-2 with Glut4-containing vesiclesInsulin induces heterologous desensitization of G-protein-coupled receptor and insulin-like growth factor I signaling by downregulating beta-arrestin-1.Identification of the APS protein as a novel insulin receptor substrateMembrane localization of 3-phosphoinositide-dependent protein kinase-1 stimulates activities of Akt and atypical protein kinase C but does not stimulate glucose transport and glycogen synthesis in 3T3-L1 adipocytes.Munc18c regulates insulin-stimulated glut4 translocation to the transverse tubules in skeletal muscle.Characterization of selective resistance to insulin signaling in the vasculature of obese Zucker (fa/fa) rats.Signaling pathways mediating insulin-stimulated glucose transport.Logical analysis of timing-dependent receptor signalling specificity: application to the insulin receptor metabolic and mitogenic signalling pathways.Beta -Arrestin 1 down-regulation after insulin treatment is associated with supersensitization of beta 2 adrenergic receptor Galpha s signaling in 3T3-L1 adipocytes.Conventional kinesin KIF5B mediates insulin-stimulated GLUT4 movements on microtubulesImpaired expression of the inducible cAMP early repressor accounts for sustained adipose CREB activity in obesity.Turning signals on and off: GLUT4 traffic in the insulin-signaling highway.Identification of an early endosomal protein regulated by phosphatidylinositol 3-kinase.Phosphatidylinositol-3,4,5-triphosphate and cellular signaling: implications for obesity and diabetes.Evidence for an insulin receptor substrate 1 independent insulin signaling pathway that mediates insulin-responsive glucose transporter (GLUT4) translocationNaturally occurring variants of the dysglycemic peptide pancreastatin: differential potencies for multiple cellular functions and structure-function correlation.Testosterone insulin-like effects: an in vitro study on the short-term metabolic effects of testosterone in human skeletal muscle cells.G alpha-q/11 protein plays a key role in insulin-induced glucose transport in 3T3-L1 adipocytes.Regulation of insulin-stimulated glucose transporter GLUT4 translocation and Akt kinase activity by ceramide.An SH2 domain-containing 5' inositolphosphatase inhibits insulin-induced GLUT4 translocation and growth factor-induced actin filament rearrangement.Adenovirus-mediated overexpression of IRS-1 interacting domains abolishes insulin-stimulated mitogenesis without affecting glucose transport in 3T3-L1 adipocytes.Insulin-mimetic and insulin-sensitizing activities of a pentacyclic triterpenoid insulin receptor activator.Hyperosmolarity reduces GLUT4 endocytosis and increases its exocytosis from a VAMP2-independent pool in l6 muscle cells.Insulin and insulin-like growth factor I receptors utilize different G protein signaling components.The tumor suppressor PTEN negatively regulates insulin signaling in 3T3-L1 adipocytes.Insulin regulates protein kinase CbetaII expression through enhanced exon inclusion in L6 skeletal muscle cells. A novel mechanism of insulin- and insulin-like growth factor-i-induced 5' splice site selection.Insulin-induced egr-1 and c-fos expression in 32D cells requires insulin receptor, Shc, and mitogen-activated protein kinase, but not insulin receptor substrate-1 and phosphatidylinositol 3-kinase activation.Functional roles of the Shc phosphotyrosine binding and Src homology 2 domains in insulin and epidermal growth factor signaling.Insulin and secretagogues differentially regulate fluid-phase pinocytosis in insulin-secreting beta-cells.Insulin second messengers.Endothelin-1-induced GLUT4 translocation is mediated via Galpha(q/11) protein and phosphatidylinositol 3-kinase in 3T3-L1 adipocytes.Insulin and insulin-like growth factor I up-regulate GLUT4 gene expression in fetal brown adipocytes, in a phosphoinositide 3-kinase-dependent manner.
P2860
Q22004172-DCD16EEC-1354-4ED9-A2EE-A2AD07B01430Q24293490-5D44714F-E5C7-420B-8398-A6A283B31B85Q24316670-FFF68FCB-B30E-400B-BB78-A8EA32D0F0DEQ24318310-D69239A3-153D-4198-90A9-60CED28C1953Q24519086-5B0B9AE1-7C45-443A-9E34-65A3A7C2CC39Q24551012-7A8C08EE-A94A-4CF0-B20D-DD8205362178Q28364553-752D9CC3-C19E-41BD-AFA3-AAA04FE94AACQ28512887-08EEF925-926B-45F3-9F33-0FD7BC559C68Q28573673-E498E107-0B72-4980-B862-6AF05D164D21Q28575441-3C8E9AC6-6FB5-4571-9670-F734FCB39D5BQ28587602-3CFA84E7-A80A-4EA1-B647-38B9C465E841Q30839834-1584CDE8-B84C-47DD-A167-2B8DA14FB8CDQ31509381-D3937CC0-8720-4139-A808-7EAF06399C9FQ33857313-550EEA24-8941-42E1-80FE-9678FC4C5BBCQ33936999-F25137F5-B900-4ED7-8475-7893D9925A12Q34438574-1665F06D-997E-47B5-B630-7E8C066660ADQ34467022-9F70599E-05C3-4239-95AD-D0C535DE2C05Q35016947-2C5EE3CB-3CAB-4CCD-A439-222B49D913A0Q35561110-9F52A971-6B9D-445F-9EBE-0CF0C4B6EE7FQ36197835-EF82426B-F536-40D5-AB15-8F8D0932CAF5Q36655286-6FFB584A-5166-4681-892D-3A39B294B483Q36702291-4449C7BC-0297-49C6-9BE0-19A5E54A9EBCQ37404030-2FC025BD-AAC7-4F2B-B82D-762C5FD8F34BQ37583609-1E5C075F-AD36-4EC2-90F3-C5512CBBA5E4Q38784181-F4DC3C55-E7A0-417C-B087-0E6CCBEA3A43Q39448110-B2020714-6ECD-4A16-BF73-5CAE0DE67D1AQ39575996-CF2AC79F-8772-4566-9A8F-9B1CA012FA05Q39610838-DC5C2183-3AF5-42ED-A0D0-A136251BB0BEQ40024191-F2311EE7-8A19-4526-A740-BAE75B079DDBQ40187486-58209E4E-7B08-46EE-B2D0-F305A728CB88Q40813496-E34B3D58-9036-4A2C-A7E3-D60C49F6C6A5Q40816727-AE7C546E-7D06-467C-B399-CAC3AEDB904EQ40884595-74E7326F-E797-425B-9EFC-90910E878DD1Q41066077-72942509-D36B-49E1-8606-B5F67D87A9D1Q41150304-54332D17-A0FF-4F42-92F4-0C09C18AA63BQ41157805-B7222242-E6D4-4F31-B2E6-99F31DF569ECQ41172024-8A383E0A-A608-4ADC-AC39-8B90EAB90FFAQ41459457-6A9351F9-AD8E-4A53-8747-7901DDAAEE8CQ41701992-1F107396-2F63-49DA-80A5-4C49FB603C0CQ42159013-6CA6A54C-77F2-4B82-8CE9-F1D218753355
P2860
Insulin-stimulated GLUT4 translocation is mediated by a divergent intracellular signaling pathway.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Insulin-stimulated GLUT4 trans ...... tracellular signaling pathway.
@en
type
label
Insulin-stimulated GLUT4 trans ...... tracellular signaling pathway.
@en
prefLabel
Insulin-stimulated GLUT4 trans ...... tracellular signaling pathway.
@en
P2093
P356
P1476
Insulin-stimulated GLUT4 trans ...... tracellular signaling pathway.
@en
P2093
P304
27991-27994
P356
10.1074/JBC.270.47.27991
P407
P577
1995-11-01T00:00:00Z