Early murine polymicrobial sepsis predominantly causes renal injury.
about
Why do they die? Comparison of selected aspects of organ injury and dysfunction in mice surviving and dying in acute abdominal sepsisNonhematopoietic β-Arrestin-1 inhibits inflammation in a murine model of polymicrobial sepsis.Location, location, location: cytokine concentrations are dependent on blood sampling site.Current Murine Models of Sepsis.Increased coagulation and suppressed generation of activated protein C in aged mice during intra-abdominal sepsis.Acute-Phase Deaths from Murine Polymicrobial Sepsis Are Characterized by Innate Immune Suppression Rather Than ExhaustionShorter Duration of Post-Operative Antibiotics for Cecal Ligation and Puncture Does Not Increase Inflammation or MortalityInnate Immune Molecule Surfactant Protein D Attenuates Sepsis-induced Acute Pancreatic Injury through Modulating Apoptosis and NF-κB-mediated Inflammation.Sepsis-3 on the Block: What Does It Mean for Preclinical Sepsis Modeling?Nonhematopoietic Peroxisome Proliferator-Activated Receptor-α Protects Against Cardiac Injury and Enhances Survival in Experimental Polymicrobial Sepsis.Imaging Apoptosis in Sepsis--A Technology We Would Die for!Role of Adenosine 1a Receptor Signaling on GFR Early After the Induction of Sepsis.
P2860
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P2860
Early murine polymicrobial sepsis predominantly causes renal injury.
description
2014 nî lūn-bûn
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name
Early murine polymicrobial sepsis predominantly causes renal injury.
@en
type
label
Early murine polymicrobial sepsis predominantly causes renal injury.
@en
prefLabel
Early murine polymicrobial sepsis predominantly causes renal injury.
@en
P2093
P2860
P1433
P1476
Early murine polymicrobial sepsis predominantly causes renal injury.
@en
P2093
Daniel G Remick
Evan L Chiswick
Florin L Craciun
Joel M Henderson
Kendra N Iskander
P2860
P304
P356
10.1097/SHK.0000000000000073
P577
2014-02-01T00:00:00Z