Differentiation-dependent autophagy controls the fate of newly synthesized N-linked glycoproteins in the colon adenocarcinoma HT-29 cell line.
about
Control of the expression and activity of the Galpha-interacting protein (GAIP) in human intestinal cellsGlucose regulates protein catabolism in ras-transformed fibroblasts through a lysosomal-dependent proteolytic pathwayReduced Warburg effect in cancer cells undergoing autophagy: steady- state 1H-MRS and real-time hyperpolarized 13C-MRS studiesG protein-coupled receptors and the regulation of autophagy.Embryonic cerebral cortical progenitors are resistant to apoptosis, but increase expression of suicide receptor DISC-complex genes and suppress autophagy following ethanol exposureThe G-protein regulator AGS3 controls an early event during macroautophagy in human intestinal HT-29 cells.Increased hyaluronan levels in HABP1/p32/gC1qR overexpressing HepG2 cells inhibit autophagic vacuolation regulating tumor potency.Programmed cell death pathways and current antitumor targets.Glucose persistence on high-mannose oligosaccharides selectively inhibits the macroautophagic sequestration of N-linked glycoproteins.Guanine nucleotide exchange on heterotrimeric Gi3 protein controls autophagic sequestration in HT-29 cells.Expression of BNIP3 correlates with hypoxia-inducible factor (HIF)-1alpha, HIF-2alpha and the androgen receptor in prostate cancer and is regulated directly by hypoxia but not androgens in cell lines.Amino acids interfere with the ERK1/2-dependent control of macroautophagy by controlling the activation of Raf-1 in human colon cancer HT-29 cells.The metabolism of sphingo(glyco)lipids is correlated with the differentiation-dependent autophagic pathway in HT-29 cells.Subcellular localization of the Galphai3 protein and G alpha interacting protein, two proteins involved in the control of macroautophagy in human colon cancer HT-29 cells.
P2860
Q28249643-9223B993-F0AD-4E8E-8F84-8B606C7A09C8Q28367246-8B45C32B-09F1-42DE-95D8-9C5B19F835E7Q28541488-37343BD2-406E-4F65-B79F-FE89B206FA77Q33847091-046BE286-A058-4846-ABAA-186390FFCF09Q34034238-FD0CEECB-4B5B-4422-9A3E-40981E637F9BQ34184261-63B08C11-41B9-4B88-8A77-BDEE956B00D0Q35213012-FF2515CF-7E89-472E-B69E-87F0B05B83C2Q37466253-A9ADB93F-43F1-400A-B832-C97E290847C3Q38316367-C51A3745-DE99-4C32-AA48-6AE0F3CB0283Q38351554-8E87124B-ACC0-479A-A41D-9F47D5F0CEDFQ40029820-272BD4BF-8768-42B5-A1DE-D7BC6EC33377Q40667540-2E9FAF29-F8CF-4332-B9FE-5CF7F02FCFA0Q41205526-A31A3553-1413-4130-BC33-D8F34CC21B52Q42992522-46B802EE-B2BC-4729-837B-B00FE217869B
P2860
Differentiation-dependent autophagy controls the fate of newly synthesized N-linked glycoproteins in the colon adenocarcinoma HT-29 cell line.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Differentiation-dependent auto ...... denocarcinoma HT-29 cell line.
@en
type
label
Differentiation-dependent auto ...... denocarcinoma HT-29 cell line.
@en
prefLabel
Differentiation-dependent auto ...... denocarcinoma HT-29 cell line.
@en
P2093
P2860
P356
P1433
P1476
Differentiation-dependent auto ...... adenocarcinoma HT-29 cell line
@en
P2093
D De Stefanis
F M Baccino
P2860
P304
P356
10.1042/BJ3090521
P407
P478
309 ( Pt 2)
P577
1995-07-01T00:00:00Z