Cigarette smoke condensate and dioxin suppress culture shock induced senescence in normal human oral keratinocytes
about
Cellular transformation by cigarette smoke extract involves alteration of glycolysis and mitochondrial function in esophageal epithelial cellsActivation of the aryl hydrocarbon receptor by TCDD inhibits senescence: a tumor promoting event?Evaluation of in vitro assays for assessing the toxicity of cigarette smoke and smokeless tobacco.2,3,7,8-tetrachlorodibenzo-p-dioxin counteracts the p53 response to a genotoxicant by upregulating expression of the metastasis marker agr2 in the hepatocarcinoma cell line HepG2.Role for the Aryl Hydrocarbon Receptor and Diverse Ligands in Oral Squamous Cell Carcinoma Migration and Tumorigenesis.Molecular alterations associated with chronic exposure to cigarette smoke and chewing tobacco in normal oral keratinocytes.Maternal human telomerase reverse transcriptase variants are associated with preterm labor and preterm premature rupture of membranes.
P2860
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P2860
Cigarette smoke condensate and dioxin suppress culture shock induced senescence in normal human oral keratinocytes
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Cigarette smoke condensate and ...... ormal human oral keratinocytes
@en
type
label
Cigarette smoke condensate and ...... ormal human oral keratinocytes
@en
prefLabel
Cigarette smoke condensate and ...... ormal human oral keratinocytes
@en
P2093
P2860
P1433
P1476
Cigarette smoke condensate and ...... ormal human oral keratinocytes
@en
P2093
C Gary Gairola
Hollie I Swanson
Joseph Valentino
R W Cameron Dingle
P2860
P304
P356
10.1016/J.ORALONCOLOGY.2006.08.008
P407
P577
2006-10-25T00:00:00Z