T-cell autonomous death induced by regeneration of inert glucocorticoid metabolites.

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Cutting Edge: De Novo Glucocorticoid Synthesis by Thymic Epithelial Cells Regulates Antigen-Specific Thymocyte Selection.

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T-cell autonomous death induced by regeneration of inert glucocorticoid metabolites.

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T-cell autonomous death induced by regeneration of inert glucocorticoid metabolites.

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T-cell autonomous death induced by regeneration of inert glucocorticoid metabolites.

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T-cell autonomous death induced by regeneration of inert glucocorticoid metabolites.

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P1476

T-cell autonomous death induced by regeneration of inert glucocorticoid metabolites.

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Andreas Villunger

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GJan Wiegers

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Holger M Reichardt

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Lourdes Rocamora-Reverte

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P2860

Glucocorticoid-induced thymocyte apoptosis is associated with endogenous endonuclease activation

Paracrine glucocorticoid activity produced by mouse thymic epithelial cells

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Disruption of the glucocorticoid receptor gene in the nervous system results in reduced anxiety

Intracellular regeneration of glucocorticoids by 11beta-hydroxysteroid dehydrogenase (11beta-HSD)-1 plays a key role in regulation of the hypothalamic-pituitary-adrenal axis: analysis of 11beta-HSD-1-deficient mice.

Positive selection of thymocytes.

Single-cell RNA sequencing reveals T helper cells synthesizing steroids de novo to contribute to immune homeostasis.

TNF suppresses acute intestinal inflammation by inducing local glucocorticoid synthesis.

Transcriptional regulation of kinases downstream of the T cell receptor: another immunomodulatory mechanism of glucocorticoids.

Glucocorticoids in T cell development and function*.

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e2948

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scholarly article

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10.1038/CDDIS.2017.344

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2017-07-20T00:00:00Z

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28726773

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5550885

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T-cell autonomous death induced by regeneration of inert glucocorticoid metabolites.

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P2860

P2860

T-cell autonomous death induced by regeneration of inert glucocorticoid metabolites.

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P2860

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