SOCS1 gene promoter methylation status is associated with in-stent restenosis after percutaneous coronary intervention.

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SOCS1 gene promoter methylation status is associated with in-stent restenosis after percutaneous coronary intervention.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

@yue

2017年論文

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2017年論文

@zh-hk

2017年論文

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2017年論文

@zh-tw

2017年论文

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2017年论文

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2017年论文

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SOCS1 gene promoter methylatio ...... taneous coronary intervention.

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SOCS1 gene promoter methylatio ...... taneous coronary intervention.

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prefLabel

SOCS1 gene promoter methylatio ...... taneous coronary intervention.

@en

P1476

SOCS1 gene promoter methylatio ...... taneous coronary intervention.

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Guoxin Tong

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Hao Pan

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Hong Li

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Jianmin Yang

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Jinyu Huang

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Lei Lai

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Liang Zhou

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Ningfu Wang

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Xianhua Ye

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P2860

Recurrent acute coronary syndrome and restenosis after percutaneous coronary intervention in a patient with idiopathic thrombocytopenic purpura: a case report and literature review

Role of JAK/STAT pathway in IL-6-induced activation of vascular smooth muscle cells

The suppressors of cytokine signalling (SOCS).

Suppressor of cytokine signaling (SOCS) genes are silenced by DNA hypermethylation and histone deacetylation and regulate response to radiotherapy in cervical cancer cells.

Inflammation and restenosis: implications for therapy.

Inflammation as a mechanism and therapeutic target for in-stent restenosis.

SOCS1: a potent and multifaceted regulator of cytokines and cell-mediated inflammation.

Aberrant methylation of suppressor of cytokine signalling-1 (SOCS-1) gene in pancreatic ductal neoplasms.

Restenosis after percutaneous angioplasty: the role of vascular inflammation.

SOCS proteins, cytokine signalling and immune regulation.

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56959-56967

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scholarly article

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10.18632/ONCOTARGET.18398

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English

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2017-06-07T00:00:00Z

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28915645

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5593616

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