Radiation-induced DNA double-strand break rejoining in human tumour cells.
about
Megabase chromatin domains involved in DNA double-strand breaks in vivop53 binding protein 1 (53BP1) is an early participant in the cellular response to DNA double-strand breaksA plausible model for the digital response of p53 to DNA damage.Ionizing radiation triggers chromatin-bound kin17 complex formation in human cells.RNA-seq profiling of a radiation resistant and radiation sensitive prostate cancer cell line highlights opposing regulation of DNA repair and targets for radiosensitization.A model of photon cell killing based on the spatio-temporal clustering of DNA damage in higher order chromatin structures.(212)Pb-radioimmunotherapy induces G(2) cell-cycle arrest and delays DNA damage repair in tumor xenografts in a model for disseminated intraperitoneal disease.Mechanism of action studies of lomaiviticin A and the monomeric lomaiviticin aglycon. Selective and potent activity toward DNA double-strand break repair-deficient cell lines.Radionuclide carriers for targeting of cancer.Continuing pursuit for ideal systemic anticancer radiotherapeutics.Development of a novel rapid assay to assess the fidelity of DNA double-strand-break repair in human tumour cells.Radiosensitivity of human tumour cells is correlated with the induction but not with the repair of DNA double-strand breaks.Sensitivity of the Giant LOop Binary LEsion (GLOBLE) cell survival model on parameters characterising dose rate effects.Heat exposure enhances radiosensitivity by depressing DNA-PK kinase activity during double strand break repair.Phosphorylation and rapid relocalization of 53BP1 to nuclear foci upon DNA damage.Relationship between p53 status and radiosensitivity in human tumour cell lines.On the role of DNA double-strand breaks in toxicity and carcinogenesis.The use of caspase inhibitors in pulsed-field gel electrophoresis may improve the estimation of radiation-induced DNA repair and apoptosis.Late G1 accumulation after 2 Gy of gamma-irradiation is related to endogenous Raf-1 protein expression and intrinsic radiosensitivity in human cellsToward intracellular targeted delivery of cancer therapeutics: progress and clinical outlook for brain tumor therapy.Combined RAF1 protein expression and p53 mutational status provides a strong predictor of cellular radiosensitivity.Threonine 68 of Chk2 is phosphorylated at sites of DNA strand breaks.Kinetics and Fidelity of the Repair of Cas9-Induced Double-Strand DNA Breaks.Effects of CRISPR/Cas9 dosage on TICAM1 and RBL gene mutation rate, embryonic development, hatchability and fry survival in channel catfish
P2860
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P2860
Radiation-induced DNA double-strand break rejoining in human tumour cells.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Radiation-induced DNA double-strand break rejoining in human tumour cells.
@en
type
label
Radiation-induced DNA double-strand break rejoining in human tumour cells.
@en
prefLabel
Radiation-induced DNA double-strand break rejoining in human tumour cells.
@en
P2093
P2860
P356
P1476
Radiation-induced DNA double-strand break rejoining in human tumour cells.
@en
P2093
McMillan TJ
Ruiz de Almodóvar JM
Valenzuela MT
Villalobos M
P2860
P2888
P304
P356
10.1038/BJC.1995.62
P407
P577
1995-02-01T00:00:00Z