Differentiation-dependent expression of E1--E4 proteins in cell lines maintaining episomes of human papillomavirus type 31b.
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Inhibition of MMP-9 expression by ritonavir or saquinavir is associated with inactivation of the AKT/Fra-1 pathway in cervical intraepithelial neoplasia cells.Induction of the human papillomavirus type 31 late promoter requires differentiation but not DNA amplificationTemporal usage of multiple promoters during the life cycle of human papillomavirus type 31b.Transactivation by the E2 protein of oncogenic human papillomavirus type 31 is not essential for early and late viral functions.Role of the E1--E4 protein in the differentiation-dependent life cycle of human papillomavirus type 31.Differentiation-dependent chromatin rearrangement coincides with activation of human papillomavirus type 31 late gene expression.Life cycle heterogeneity in animal models of human papillomavirus-associated diseaseHuman papillomavirus type 31 E5 protein supports cell cycle progression and activates late viral functions upon epithelial differentiation.Characterization of late gene transcripts expressed during vegetative replication of human papillomavirus type 31bFunctional analysis of the human papillomavirus type 16 E1=E4 protein provides a mechanism for in vivo and in vitro keratin filament reorganization.In vitro synthesis of oncogenic human papillomaviruses requires episomal genomes for differentiation-dependent late expression.Regulation of human papillomavirus type 31 gene expression during the differentiation-dependent life cycle through histone modifications and transcription factor bindingHuman papillomavirus 18 E1^E4 protein interacts with cyclin A/CDK 2 through an RXL motif.Activation of papillomavirus late gene transcription and genome amplification upon differentiation in semisolid medium is coincident with expression of involucrin and transglutaminase but not keratin-10.Modulation of the cell division cycle by human papillomavirus type 18 E4.Human papillomavirus type 31b infection of human keratinocytes and the onset of early transcription.Human papillomavirus type 31b late gene expression is regulated through protein kinase C-mediated changes in RNA processing.Mutational analysis of the human papillomavirus type 16 E1--E4 protein shows that the C terminus is dispensable for keratin cytoskeleton association but is involved in inducing disruption of the keratin filaments.Induction of human papillomavirus type 18 late gene expression and genomic amplification in organotypic cultures from transfected DNA templates.mRNA splicing regulates human papillomavirus type 11 E1 protein production and DNA replication.Role for Wee1 in inhibition of G2-to-M transition through the cooperation of distinct human papillomavirus type 1 E4 proteins.SETD2-dependent H3K36me3 plays a critical role in epigenetic regulation of the HPV31 life cycle
P2860
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P2860
Differentiation-dependent expression of E1--E4 proteins in cell lines maintaining episomes of human papillomavirus type 31b.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Differentiation-dependent expr ...... human papillomavirus type 31b.
@en
type
label
Differentiation-dependent expr ...... human papillomavirus type 31b.
@en
prefLabel
Differentiation-dependent expr ...... human papillomavirus type 31b.
@en
P1433
P1476
Differentiation-dependent expr ...... human papillomavirus type 31b.
@en
P2093
P304
P356
10.1016/S0042-6822(95)80088-3
P407
P577
1995-01-01T00:00:00Z