about
Passage of parasites across the blood-brain barrierCoadministration of indomethacin and minocycline attenuates established paclitaxel-induced neuropathic thermal hyperalgesia: Involvement of cannabinoid CB1 receptorsEnhancement of antinociception by coadministration of minocycline and a non-steroidal anti-inflammatory drug indomethacin in naïve mice and murine models of LPS-induced thermal hyperalgesia and monoarthritisIdentification of stage biomarkers for human African trypanosomiasis.Paclitaxel-induced hyposensitivity to nociceptive chemical stimulation in mice can be prevented by treatment with minocycline.Gait analysis of C57BL/6 mice with complete Freund's adjuvant-induced arthritis using the CatWalk systemCOL-3, a chemically modified tetracycline, inhibits lipopolysaccharide-induced microglia activation and cytokine expression in the brain.Astrocyte activation in the anterior cingulate cortex and altered glutamatergic gene expression during paclitaxel-induced neuropathic pain in mice.Migration of African trypanosomes across the blood-brain barrier.Gene expression profile of sodium channel subunits in the anterior cingulate cortex during experimental paclitaxel-induced neuropathic pain in mice.Mechanisms of CNS invasion and damage by parasites.Distinct Toll-like receptor signals regulate cerebral parasite load and interferon α/β and tumor necrosis factor α-dependent T-cell infiltration in the brains of Trypanosoma brucei-infected mice.Comprehensive analysis of the GABAergic system gene expression profile in the anterior cingulate cortex of mice with Paclitaxel-induced neuropathic pain.Minocycline impedes African trypanosome invasion of the brain in a murine model.Matrix metalloproteinase inhibitor COL-3 prevents the development of paclitaxel-induced hyperalgesia in mice.CD200 receptors are differentially expressed and modulated by minocycline in the brain during Trypanosoma brucei infection.Systemic lipopolysaccharide (LPS)-induced microglial activation results in different temporal reduction of CD200 and CD200 receptor gene expression in the brain.Mechanisms involved in morphine-induced activation of synaptosomal Na+,K+-ATPase.Distinguishing subgroups among μ-opioid receptor agonists using Na(+),K(+)-ATPase as an effector mechanism.Role of Na(+), K(+)-ATPase in morphine-induced antinociception.Expression and role of CXCL10 during the encephalitic stage of experimental and clinical African trypanosomiasis.Differential invasion of Trypanosoma brucei brucei and lymphocytes into the brain of C57BL/6 and 129Sv/Ev mice.Suramin and minocycline treatment of experimental African trypanososmiasis at an early stage of parasite brain invasion.Treatment of African trypanosomiasis with cordycepin and adenosine deaminase inhibitors in a mouse model.Assessment of weight bearing changes and pharmacological antinociception in mice with LPS-induced monoarthritis using the Catwalk gait analysis system.Changes in morphine-induced activation of cerebral Na(+),K(+)-ATPase during morphine tolerance: biochemical and behavioral consequences.The anticonvulsant enaminone E139 attenuates paclitaxel-induced neuropathic pain in rodents.Targeting the Endocannabinoid System for Prevention or Treatment of Chemotherapy-Induced Neuropathic Pain: Studies in Animal ModelsGene expression profile of sodium channel subunits in the anterior cingulate cortex during experimental paclitaxel-induced neuropathic pain in miceGene expression profile of sodium channel subunits in the anterior cingulate cortex during experimental paclitaxel-induced neuropathic pain in miceAstrocyte activation in the anterior cingulate cortex and altered glutamatergic gene expression during paclitaxel-induced neuropathic pain in miceAstrocyte activation in the anterior cingulate cortex and altered glutamatergic gene expression during paclitaxel-induced neuropathic pain in miceAstrocyte activation in the anterior cingulate cortex and altered glutamatergic gene expression during paclitaxel-induced neuropathic pain in miceCorrigendum to “Assessment of weight bearing changes and pharmacological antinociception in mice with LPS-induced monoarthritis using the Catwalk gait analysis system” [Life Sciences 85 (2009) 462–469]Cerebral vessel laminins and IFN-γ define Trypanosoma brucei brucei penetration of the blood-brain barrierβ-Caryophyllene, a CB2-Receptor-Selective Phytocannabinoid, Suppresses Mechanical Allodynia in a Mouse Model of Antiretroviral-Induced Neuropathic Pain
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description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Willias Masocha
@ast
Willias Masocha
@en
Willias Masocha
@es
Willias Masocha
@nl
Willias Masocha
@sl
type
label
Willias Masocha
@ast
Willias Masocha
@en
Willias Masocha
@es
Willias Masocha
@nl
Willias Masocha
@sl
prefLabel
Willias Masocha
@ast
Willias Masocha
@en
Willias Masocha
@es
Willias Masocha
@nl
Willias Masocha
@sl
P106
P1153
6506662720
P21
P31
P496
0000-0003-3132-5935