Cell plasticity in wound healing: paracrine factors of M1/ M2 polarized macrophages influence the phenotypical state of dermal fibroblasts. - Wikidata - awgldk - TriplyDB

Cell plasticity in wound healing: paracrine factors of M1/ M2 polarized macrophages influence the phenotypical state of dermal fibroblasts.

Cell plasticity in wound healing: paracrine factors of M1/ M2 polarized macrophages influence the phenotypical state of dermal fibroblasts.

http://www.wikidata.org/entity/Q41382922

about

Glioblastoma: a pathogenic crosstalk between tumor cells and pericytes Complement Activation and STAT4 Expression Are Associated with Early Inflammation in Diabetic Wounds.Water-soluble egg membrane enhances the immunoactivating properties of an Aloe vera-based extract of Nerium oleander leaves.Antioxidant, anti-inflammatory, anti-apoptotic, and skin regenerative properties of an Aloe vera-based extract of Nerium oleander leaves (nae-8(®)).Clinical challenges of chronic wounds: searching for an optimal animal model to recapitulate their complexity TPL2 kinase regulates the inflammatory milieu of the myeloma niche Induction of m2 polarization in primary culture liver macrophages from rats with acute pancreatitis MicroRNAs Control Macrophage Formation and Activation: The Inflammatory Link between Obesity and Cardiovascular Diseases Specific sizes of hyaluronan oligosaccharides stimulate fibroblast migration and excisional wound repair.Fibrosis, vascular activation, and immune abnormalities resembling systemic sclerosis in bleomycin-treated Fli-1-haploinsufficient mice Quantitative Characterization of Collagen in the Fibrotic Capsule Surrounding Implanted Polymeric Microparticles through Second Harmonic Generation Imaging.Mesenchymal stem cell-derived inflammatory fibroblasts promote monocyte transition into myeloid fibroblasts via an IL-6-dependent mechanism in the aging mouse heart.Forkhead Box O1 Regulates Macrophage Polarization Following Staphylococcus aureus Infection: Experimental Murine Data and Review of the Literature.Increased Infiltration of Extra-Cardiac Cells in Myxomatous Valve Disease.Interleukin-6 stimulates Akt and p38 MAPK phosphorylation and fibroblast migration in non-diabetic but not diabetic mice Early infiltration of p40IL12(+)CCR7(+)CD11b(+) cells is critical for fibrosis development.A quorum-sensing signal promotes host tolerance training through HDAC1-mediated epigenetic reprogramming.New approaches to modulating idiopathic pulmonary fibrosis.Combining tissue repair and tissue engineering; bioactivating implantable cell-free vascular scaffolds.Bidirectional crosstalk via IL-6, PGE2 and PGD2 between murine myofibroblasts and alternatively activated macrophages enhances anti-inflammatory phenotype in both cells.Alternatively activated macrophages derived from THP-1 cells promote the fibrogenic activities of human dermal fibroblasts.Macrophage-based therapeutic strategies in regenerative medicine.Angiopoietin-like 4 induces a β-catenin-mediated upregulation of ID3 in fibroblasts to reduce scar collagen expression.Recent advances in biomaterials for the treatment of diabetic foot ulcers.Therapeutic assessment of mesenchymal stem cells delivered within a PEGylated fibrin gel following an ischemic injury.Activated macrophages control human adipocyte mitochondrial bioenergetics via secreted factors IL-10 improves cardiac remodeling after myocardial infarction by stimulating M2 macrophage polarization and fibroblast activation.Drug delivery and tissue engineering to promote wound healing in the immunocompromised host: Current challenges and future directions.In vivo response to decellularized mesothelium scaffolds.Zika Virus Infection Preferentially Counterbalances Human Peripheral Monocyte and/or NK Cell Activity.Biomaterial-driven in situ cardiovascular tissue engineering-a multi-disciplinary perspective.Allogeneic platelet-rich plasma affects monocyte differentiation to dendritic cells causing an anti-inflammatory microenvironment, putatively fostering wound healing.Inflammatory licensed equine MSCs are chondroprotective and exhibit enhanced immunomodulation in an inflammatory environment.Black, White, and Gray: Macrophages in Skin Repair and Disease

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P2860

Cell plasticity in wound healing: paracrine factors of M1/ M2 polarized macrophages influence the phenotypical state of dermal fibroblasts.

http://www.wikidata.org/entity/Q41382922

description

2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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2013年论文

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2013年论文

@zh-cn

name

Cell plasticity in wound heali ...... l state of dermal fibroblasts.

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type

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Cell plasticity in wound heali ...... l state of dermal fibroblasts.

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Cell plasticity in wound heali ...... l state of dermal fibroblasts.

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Cell Communication and Signaling

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Cell plasticity in wound heali ...... l state of dermal fibroblasts.

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P2093

Diana Ta Ploeger

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Jasper A Koerts

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Martin Schipper

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Nynke A Hosper

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Ruud A Bank

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Saskia de Rond

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P2860

Cellular and molecular mechanisms of fibrosis

Exploring the full spectrum of macrophage activation

Fibroblasts and monocyte macrophages contract and degrade three-dimensional collagen gels in extended co-culture

Collaborative interactions between neutrophil elastase and metalloproteinases in extracellular matrix degradation in three-dimensional collagen gels

Alternative activation of macrophages

CSF-1 as a regulator of macrophage activation and immune responses

Transcriptional profiling of the human monocyte-to-macrophage differentiation and polarization: new molecules and patterns of gene expression

Monocyte and macrophage heterogeneity

Wound repair and regeneration

Cutaneous wound healing

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10.1186/1478-811X-11-29

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